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Cell line · CVCL_0332

Papers156

2 more articles give this line's RRID in its Methods without naming the line nearby, so they are not listed.

1 to 20 of 156

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PaperJournalYearHow we knowRetractedSource
VCP-Regulated LMF2 Stabilizes the Cholesterol Biosynthesis Enzyme HMGCS1 to Drive Cancer Progression and Confer Capecitabine Resistance in Triple-Negative Breast Cancer.Adv Sci (Weinh)2026Its Methods give the RRID and name the linePubMed
The role of autoproteolysis and mitoribosomal proteins in the regulation of the mitochondrial LACTB tumor suppressor.EMBO Rep2026Its Methods give the RRID and name the linePubMed
Lysyl oxidase inhibition disrupts mitochondrial homeostasis to create vulnerability to ferroptosis in TNBC.Cell Rep Med2026Its Methods give the RRID and name the linePubMed
Role of orphan G protein-coupled receptor GPR52 in breast cancer cell multicellular organization and collective invasion.Br J Cancer2026Its Methods give the RRID and name the linePubMed
Intratumoral Hypoxia Triggers Mitochondrial BHLHE40 ROS Sensing Pathway to Promote Radioresistance in Triple-Negative Breast Cancer.Adv Sci (Weinh)2026Its Methods give the RRID and name the linePubMed
ENO2 sustains cancer stemness and metastatic competence through a phosphoenolpyruvate-dependent metabolic axis in triple-negative breast cancer.Cell Death Dis2026Its Methods give the RRID and name the linePubMed
Distinct metabolic dependency on BCAA defines cancer stemness and malignancy in human triple-negative breast cancer.Cell Rep2026Its Methods give the RRID and name the linePubMed
UBE2L6 promotes invasion and metastasis of triple-negative breast cancer by enhancing autophagy through STK38 ISGylation.Cell Mol Life Sci2026Its Methods give the RRID and name the linePubMed
Covalent Inhibition of SHMT2 by Gambogic Acid Induces Ferroptosis Through Mitochondrial Collapse in Triple-Negative Breast Cancer.Adv Sci (Weinh)2026Its Methods give the RRID and name the linePubMed
Insulin-like growth factor 1 receptor regulates breast cancer cell adhesion through beta-1 integrin.Front Endocrinol (Lausanne)2026Its Methods give the RRID and name the linePubMed
Targeting DNA Polymerase Epsilon Induces Tumor Clearance and Activates an NF-κB-Mediated Inflammatory Response in Triple-Negative Breast Cancer.Cancer Res2026Its Methods give the RRID and name the linePubMed
Integrative Genomic Analysis Identifies MAGT1 as a Key Regulator of Proliferation and Poor Prognosis in Breast Cancer.Hum Mutat2026Its Methods give the RRID and name the linePubMed
Targeting the noncanonical function of metabolic enzyme PHGDH in driving PD-L1 expression and cancer immune evasion.Cell Rep Med2026Its Methods give the RRID and name the linePubMed
Comprehensive single-cell metabolic profiling identifies targets to sensitize triple-negative breast cancer to chemo-immunotherapy.Cell Rep Med2026Its Methods give the RRID and name the linePubMed
Discovery of novel circular RNAs of the apoptosis-related BAX gene in breast cancer, by combining nanopore and next-generation sequencing.Mol Genet Genomics2026Its Methods give the RRID and name the linePubMed
CircNSD2 promotes metastasis and immune escape by increasing the USP10/SRSF6-mediated alternative splicing of TPM1 in TNBC.Mol Cancer2026Its Methods give the RRID and name the linePubMed
COMP-PMEPA1 axis promotes epithelial-to-mesenchymal transition in breast cancer cells.Mol Oncol2026Its Methods give the RRID and name the linePubMed
Discovery and Evaluation of Biomarkers for Triple-Negative Breast Cancer Subtypes Uncovers Patient Stratification and Targeted Therapeutic Strategies.Cancer Res2026Its Methods give the RRID and name the linePubMed
Cyclic di-GMP suppresses cancer metastasis by targeting proteasome 26S subunit non-ATPase 3 independently of STING.Signal Transduct Target Ther2026Its Methods give the RRID and name the linePubMed
ZSTK3744, a Novel Aryl Hydrocarbon Receptor Agonist, Exhibits Efficacy against Chemotherapy-Resistant Triple-Negative Breast Cancer.Cancer Res Commun2026Its Methods give the RRID and name the linePubMed

cell-lines-2026-11-14-0.1.0

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  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
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  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

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