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Cell line · CVCL_0374

Papers71

1 more article gives this line's RRID in its Methods without naming the line nearby, so it is not listed.

PaperJournalYearHow we knowRetractedSource
Greatwall kinase regulates acute myeloid leukaemia cell division through a non-canonical mechanism.EMBO Rep2026Its Methods give the RRID and name the linePubMed
PD-L1 Expression in Acute Myeloid Leukemia Cells: Associations With Cell Metabolism.J Immunol Res2026Its Methods give the RRID and name the linePubMed
Integrative multi-omics, machine learning, and experimental validation reveal that TPM1 suppresses M2 macrophage polarization and enhances chemosensitivity in acute myeloid leukemia.Cell Biol Toxicol2026Its Methods give the RRID and name the linePubMed
m6A Modification of ATOX1 Inhibits Acute Myeloid Leukemia Progression by Promoting Cuproptosis.Cancer Res Commun2026Its Methods give the RRID and name the linePubMed
ΔNp73 isoform defines a TP53-mutant-like poor-risk subgroup of acute myeloid leukemia.Cell Rep Med2026Its Methods give the RRID and name the linePubMed
Schlafen 12 Modulation and Targeting in Acute Myeloid Leukemia.Cancer Res Commun2025Its Methods give the RRID and name the linePubMed
The bispecific innate cell engager AFM28 eliminates CD123+ leukemic stem and progenitor cells in AML and MDS.Nat Commun2025Its Methods give the RRID and name the linePubMed
Energy Deficiency-Induced ATG4B Nuclear Translocation Inhibits PRMT1-Mediated DNA Repair and Promotes Leukemia Progression.Adv Sci (Weinh)2025Its Methods give the RRID and name the linePubMed
Catalytic Inhibition of KAT6/KAT7 Enhances the Efficacy and Overcomes Primary and Acquired Resistance to Menin Inhibitors in MLL Leukemia.Cancer Discov2025Its Methods give the RRID and name the linePubMed
CD97-directed CAR-T cells with enhanced persistence eradicate acute myeloid leukemia in diverse xenograft models.Cell Rep Med2025Its Methods give the RRID and name the linePubMed
Aberrant expression of human endogenous retrovirus K9-derived elements is associated with better clinical outcome of acute myelocytic leukemia.Retrovirology2025Its Methods give the RRID and name the linePubMed
EZH1/EZH2 inhibition enhances adoptive T cell immunotherapy against multiple cancer models.Cancer Cell2025Its Methods give the RRID and name the linePubMed
Zelenirstat Inhibits N-Myristoyltransferases to Disrupt Src Family Kinase Signaling and Oxidative Phosphorylation, Killing Acute Myeloid Leukemia Cells.Mol Cancer Ther2025Its Methods give the RRID and name the linePubMed
B7H6 is the predominant activating ligand driving natural killer cell-mediated killing in patients with liquid tumours: evidence from clinical, in silico, in vitro, and in vivo studies.EBioMedicine2024Its Methods give the RRID and name the linePubMed
Osimertinib Covalently Binds to CD34 and Eliminates Myeloid Leukemia Stem/Progenitor Cells.Cancer Res2024Its Methods give the RRID and name the linePubMed
Myeloid/natural killer (NK) cell precursor acute leukemia as a distinct leukemia type.Sci Adv2023Its Methods give the RRID and name the linePubMed
TSC22D3 as an immune-related prognostic biomarker for acute myeloid leukemia.iScience2023Its Methods give the RRID and name the linePubMed
Targeting BET Proteins Downregulates miR-33a To Promote Synergy with PIM Inhibitors in CMML.Clin Cancer Res2023Its Methods give the RRID and name the linePubMed
Choosing the Right Cell Line for Acute Myeloid Leukemia (AML) Research.Int J Mol Sci2023Cellosaurus cites itPubMed
Transcriptionally imprinted glycomic signatures of acute myeloid leukemia.Cell Biosci2023Its Methods give the RRID and name the linePubMed

cell-lines-2026-11-14-0.1.0

Sources 4

  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
Each source's terms
  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

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