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Cell line · CVCL_0399

Papers153

PaperJournalYearHow we knowRetractedSource
NDFIP2 Suppresses Colorectal Cancer Metastasis Through DDX3X-Dependent R-Loop Remodeling and GNG13 Transcriptional Activation.Adv Sci (Weinh)2026Its Methods give the RRID and name the linePubMed
MZF1 Inhibits TGFβ-1/SMAD Signaling by Transcriptionally Activating SnoN to Augment Tumorigenic Potential in Colon Cancer.Adv Sci (Weinh)2026Its Methods give the RRID and name the linePubMed
The Mechanism Underlying Inhibition of Colorectal Cancer Invasion by Quxie Capsule Through CRK-RAC Signaling Pathway Regulation.Integr Cancer Ther2026Its Methods give the RRID and name the linePubMed
Allele-Specific Mechanisms Guide Salvage Therapy to Overcome Daraxonrasib Resistance in Pancreatic Cancer.Cancer Res2026Its Methods give the RRID and name the linePubMed
CLK3 regulates tumor angiogenesis by modulating HIF1α ubiquitination.Cancer Gene Ther2026Its Methods give the RRID and name the linePubMed
Overcoming Daraxonrasib Resistance: Allele-Specific Mechanisms Guide Salvage Therapy in Pancreatic Cancer.bioRxiv2026Its Methods give the RRID and name the linePubMed
HP1γ promotes the progression of colorectal cancer through interaction with RBPJ of the Notch signaling pathway.Cell Biol Toxicol2026Its Methods give the RRID and name the linePubMed
FUT2-Mediated Fucosylation of OLFM4 Promotes Colon Cancer Cell Differentiation.Can J Gastroenterol Hepatol2026Its Methods give the RRID and name the linePubMed
PRKN mediates the ubiquitination of SCAF8 to reduce the mRNA stability of KLF5 and its transcriptional activation of EFNA3 in colorectal cancer.Cell Mol Biol Lett2026Its Methods give the RRID and name the linePubMed
Soluble Uric Acid Drives CD8+ T-cell Exhaustion by Inducing KSR1-Mediated MAPK Hyperactivation.Cancer Res2026Its Methods give the RRID and name the linePubMed
LINE1 RNA demethylation sensitizes cancer cells to PARPi through global chromatin remodeling.Genome Biol2026Its Methods give the RRID and name the linePubMed
Role of Ca2+-Dependent Epithelial-Mesenchymal Transition in Malignant Progression of Colorectal Cancer: Special Focus on REG Iα/EDNRB.Cancer Med2026Its Methods give the RRID and name the linePubMed
A convergent uPAR-positive tumor ecosystem creates broad vulnerability to CAR T cell therapy.Cell2026Its Methods give the RRID and name the linePubMed
Visceral and subcutaneous adipose stem cells modulate colorectal cancer cell progression: direct and indirect contact distinctly accelerate tumor aggressiveness.Mol Med2026Its Methods give the RRID and name the linePubMed
ETV4 Promotes Colorectal Cancer Progression by Reprogramming Asparagine Metabolism to Remodel the Stromal Microenvironment.Adv Sci (Weinh)2026Its Methods give the RRID and name the linePubMed
METTL3 Methylation Induces Decay of Endogenous Retroelement Transcripts to Promote Tumor Immune Evasion.Cancer Res2026Its Methods give the RRID and name the linePubMed
Cyclic di-GMP suppresses cancer metastasis by targeting proteasome 26S subunit non-ATPase 3 independently of STING.Signal Transduct Target Ther2026Its Methods give the RRID and name the linePubMed
Thimerosal Inhibits Tumor Malignant Progression through Direct Action and Enhancing the Efficacy of PD-1-Based Immunotherapy.Oncol Res2026Its Methods give the RRID and name the linePubMed
The dysadherin/carbonic anhydrase 9 axis shapes an acidic tumor microenvironment to promote colorectal cancer progression.Signal Transduct Target Ther2026Its Methods give the RRID and name the linePubMed
Targeting Peptostreptococcus anaerobius with an Iron-Based Nanozyme Reverses Ferroptosis Resistance and Enhances Antitumor Immunity in Colorectal Cancer.Adv Sci (Weinh)2026Its Methods give the RRID and name the linePubMed

cell-lines-2026-11-14-0.1.0

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  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
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  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

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