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Cell line · CVCL_0505

Papers42

PaperJournalYearHow we knowRetractedSource
Auranofin Potentiates Cisplatin Response in the Setting of Context-Dependent NOTCH-Associated Signaling States in Endometrial Cancer.Cancer Res Commun2026Its Methods give the RRID and name the linePubMed
p190A/ARHGAP35 and p190B/ARHGAP5 proteins in endometrial cancer: a novel cancer-relevant paralog interplay.Mol Oncol2026Its Methods give the RRID and name the linePubMed
HRAS promotes mutant NRAS-driven transformation with codon and allele specificity.Sci Signal2026Its Methods give the RRID and name the linePubMed
Meditope-Enabled Chimeric Antigen Receptors Facilitate Plug-and-Play Control of T Cells.Cancer Immunol Res2026Its Methods give the RRID and name the linePubMed
Targeting Steroid-Metabolizing Enzymes with 15β-Substituted Estrone Analogues: Dual Discovery of AKR1C2/17β-HSD1 Inhibitors and a Fluorescent 17β-HSD1 Ligand.Cancers (Basel)2026Its Methods give the RRID and name the linePubMed
NSD1-Mediated PPARγ Methylation Enhances PTEN Activity to Suppress Glycolysis and Tumor Progression in Endometrial Cancer.Cancer Res2026Its Methods give the RRID and name the linePubMed
Microsatellite Instable Cancer Cells Acquire On-target Resistance Mutations to WRN Helicase Inhibitors.Mol Cancer Ther2026Its Methods give the RRID and name the linePubMed
The Impact of JAK1 Pathogenic Variants and MHC-I Expression on Response to Immune Checkpoint Inhibition in Endometrial Cancer.Clin Cancer Res2025Its Methods give the RRID and name the linePubMed
Wild-type RAS signaling is an essential therapeutic target in RAS-mutated cancers.Sci Signal2025Its Methods give the RRID and name the linePubMed
Pathway metabolite ratios reveal distinctive glutamine metabolism in a subset of proliferating cells.Mol Syst Biol2025Its Methods give the RRID and name the linePubMed
Functional Profiling of p53 and RB Cell Cycle Regulatory Proficiency Suggests Mechanism-Driven Molecular Stratification in Endometrial Carcinoma.Cancer Res Commun2025Its Methods give the RRID and name the linePubMed
Multi-omic analyses identified SFRP4 as a novel biomarker in abnormal uterine bleeding with ovulatory dysfunction.Heliyon2024Its Methods give the RRID and name the linePubMed
Pyroptosis-associated genes and tumor immune response in endometrial cancer.Discov Oncol2024Its Methods give the RRID and name the linePubMed
Pre-receptor regulation of 11-oxyandrogens differs between normal and cancerous endometrium and across endometrial cancer grades and molecular subtypes.Front Endocrinol (Lausanne)2024Its Methods give the RRID and name the linePubMed
PELP1 inhibition by SMIP34 reduces endometrial cancer progression via attenuation of ribosomal biogenesis.Mol Oncol2024Its Methods give the RRID and name the linePubMed
Inhibition of a lower potency target drives the anticancer activity of a clinical p38 inhibitor.Cell Chem Biol2023Its Methods give the RRID and name the linePubMed
Pan-cancer proteomic map of 949 human cell lines.Cancer Cell2022Cellosaurus cites itPubMed
Physiological Concentrations of Cimicifuga racemosa Extract Do Not Affect Expression of Genes Involved in Estrogen Biosynthesis and Action in Endometrial and Ovarian Cell Lines.Biomolecules2022Its Methods give the RRID and name the linePubMed
In the Model Cell Lines of Moderately and Poorly Differentiated Endometrial Carcinoma, Estrogens Can Be Formed via the Sulfatase Pathway.Front Mol Biosci2021Its Methods give the RRID and name the linePubMed
MET Mutation Is a Potential Therapeutic Target for Advanced Endometrial Cancer.Cancers (Basel)2021Its Methods give the RRID and name the linePubMed

cell-lines-2026-11-14-0.1.0

Sources 4

  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
Each source's terms
  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

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