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Cell line · CVCL_0524

Papers25

1 more article gives this line's RRID in its Methods without naming the line nearby, so it is not listed.

PaperJournalYearHow we knowRetractedSource
RAS inhibition limits oncogenic signaling and tumorigenesis in neuroblastoma regardless of RAS mutational statusSci Rep2026Its Methods give the RRID and name the linePubMed
PRMT5-mediated intron retention triggers innate and adaptive immunity against cancer.EMBO Mol Med2026Its Methods give the RRID and name the linePubMed
Hypoxia Promotes an Adrenergic to Mesenchymal Transcriptional Program Transition in Neuroblastoma.Clin Transl Sci2026Its Methods give the RRID and name the linePubMed
YAP1 Enhances Mesenchymal-Type Gene Expression in Human Adrenergic-Type Neuroblastoma Cells.Cancers (Basel)2026Its Methods give the RRID and name the linePubMed
Dual targeting of AMRC12 and Malassezia globosa disrupts MYC liquid condensates-driven nuclear pore complex biogenesis in neuroblastoma.Theranostics2026Its Methods give the RRID and name the linePubMed
Activity of Direct KRAS(G12C) Inhibitors in Preclinical Models of Pediatric Cancer.Mol Cancer Ther2026Its Methods give the RRID and name the linePubMed
Extrachromosomal DNA-Driven Oncogene Dosage Heterogeneity Promotes Rapid Adaptation to Therapy in MYCN-Amplified Cancers.Cancer Discov2025Its Methods give the RRID and name the linePubMed
PD-L1 Expression is Mediated by microRNA Processing, Wnt/β-Catenin Signaling, and Chemotherapy in Wilms Tumor.Pediatr Blood Cancer2025Its Methods give the RRID and name the linePubMed
Serially Quantifying TERT Rearrangement Breakpoints in ctDNA Enables Minimal Residual Disease Monitoring in Patients with Neuroblastoma.Cancer Res Commun2025Its Methods give the RRID and name the linePubMed
Sustained cancer-relevant alternative RNA splicing events driven by PRMT5 in high-risk neuroblastoma.Mol Oncol2025Its Methods give the RRID and name the linePubMed
METTL3/MYCN cooperation drives neural crest differentiation and provides therapeutic vulnerability in neuroblastoma.EMBO J2024Its Methods give the RRID and name the linePubMed
Combined inhibition of histone methyltransferases EZH2 and DOT1L is an effective therapy for neuroblastoma.Cancer Med2024Its Methods give the RRID and name the linePubMed
Characterizing Relationships between T-cell Inflammation and Outcomes in Patients with High-Risk Neuroblastoma According to Mesenchymal and Adrenergic Signatures.Cancer Res Commun2024Its Methods give the RRID and name the linePubMed
Leveraging a self-cleaving peptide for tailored control in proximity labeling proteomics.Cell Rep Methods2024Its Methods give the RRID and name the linePubMed
Inhibition of mitochondrial translocase SLC25A5 and histone deacetylation is an effective combination therapy in neuroblastoma.Int J Cancer2023Its Methods give the RRID and name the linePubMed
Severe congenital myasthenic syndromes caused by agrin mutations affecting secretion by motoneurons.Acta Neuropathol2022Its Methods give the RRID and name the linePubMed
Expanding the phenotypic variability of MORC2 gene mutations: From Charcot-Marie-Tooth disease to late-onset pure motor neuropathy.Hum Mutat2022Its Methods give the RRID and name the linePubMed
MYCN-driven fatty acid uptake is a metabolic vulnerability in neuroblastoma.Nat Commun2022Its Methods give the RRID and name the linePubMed
Inhibiting PHGDH with NCT-503 reroutes glucose-derived carbons into the TCA cycle, independently of its on-target effect.J Enzyme Inhib Med Chem2021Its Methods give the RRID and name the linePubMed
Interplay between MycN and c-Myc regulates radioresistance and cancer stem cell phenotype in neuroblastoma upon glutamine deprivation.Theranostics2020Its Methods give the RRID and name the linePubMed

cell-lines-2026-11-14-0.1.0

Sources 4

  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
Each source's terms
  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

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