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Cell line · CVCL_1345

Papers68

1 more article gives this line's RRID in its Methods without naming the line nearby, so it is not listed.

PaperJournalYearHow we knowRetractedSource
Kallikrein-Related Peptidase 5 Is Associated with Tumor Progression, KRT19 Expression, and Distinct Molecular Programs in High-Grade Serous Ovarian Cancer.Cancers (Basel)2026Its Methods give the RRID and name the linePubMed
Targeting Steroid-Metabolizing Enzymes with 15β-Substituted Estrone Analogues: Dual Discovery of AKR1C2/17β-HSD1 Inhibitors and a Fluorescent 17β-HSD1 Ligand.Cancers (Basel)2026Its Methods give the RRID and name the linePubMed
CCDC80 suppresses high-grade serous ovarian cancer migration via negative regulation of B7-H3.Mol Oncol2026Its Methods give the RRID and name the linePubMed
Using targeted therapy to promote a pro-inflammatory tumour microenvironment and anti-tumour immune response in high grade serous ovarian cancer.Br J Cancer2026Its Methods give the RRID and name the linePubMed
BMAL2 is a druggable target for ovarian clear cell carcinoma (OCCC).EMBO Mol Med2026Its Methods give the RRID and name the linePubMed
A convergent uPAR-positive tumor ecosystem creates broad vulnerability to CAR T cell therapy.Cell2026Its Methods give the RRID and name the linePubMed
Tumor-Infiltrating Nociceptor Neurons in Ovarian Cancer Treatment Resistance.Adv Biol (Weinh)2026Its Methods give the RRID and name the linePubMed
Metabolome analysis identified exogenous cholesterol within lipid rafts that activate the Akt/mTOR signaling pathway in epithelial ovarian cancer.Int J Cancer2026Its Methods give the RRID and name the linePubMed
Keratin 19 as a prognostic marker and contributing factor of metastasis and chemoresistance in high-grade serous ovarian cancer.Mol Oncol2026Its Methods give the RRID and name the linePubMed
Mesothelial cells promote peritoneal invasion and metastasis of ascites-derived ovarian cancer cells through spheroid formation.Sci Adv2026Its Methods give the RRID and name the linePubMed
PARG regulates the proteasomal degradation of TARG1.Cell Rep2026Its Methods give the RRID and name the linePubMed
Repurposing of ivacaftor shows potential to treat ROR1 expressing high-grade serous ovarian cancer.Ther Adv Med Oncol2026Its Methods give the RRID and name the linePubMed
Dynamic and Ongoing De Novo L1 Retrotransposition Contributes to Genome Plasticity and Intrapatient Heterogeneity in Ovarian Cancer.Cancer Res2026Its Methods give the RRID and name the linePubMed
PLK1 or WEE1 inhibition targets homologous recombination repair proficiency in BRCA1/2 wild-type high-grade serous ovarian cancer.Cell Death Dis2025Its Methods give the RRID and name the linePubMed
SLC25A45 is required for mitochondrial uptake of methylated amino acids and de novo carnitine biosynthesis.Mol Cell2025Its Methods give the RRID and name the linePubMed
Defining the Antitumor Mechanism of Action of a Clinical-stage Compound as a Selective Degrader of the Nuclear Pore Complex.Cancer Discov2025Its Methods give the RRID and name the linePubMed
Targeting of PTP4A3 overexpression sensitises HGSOC cells towards chemotherapeutic drugs.Mol Oncol2025Its Methods give the RRID and name the linePubMed
Tumor clusters with divergent inflammation and human retroelement expression determine the clinical outcome of patients with serous ovarian cancer.Mol Oncol2025Its Methods give the RRID and name the linePubMed
Targeting SUMOylation in ovarian cancer: Sensitivity, resistance, and the role of MYC.iScience2025Its Methods give the RRID and name the linePubMed
Systematic profiling of cancer-fibroblast interactions reveals drug combinations in ovarian cancer.Mol Oncol2025Its Methods give the RRID and name the linePubMed

cell-lines-2026-11-14-0.1.0

Sources 4

  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
Each source's terms
  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

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