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Cell line · CVCL_1465

Papers34

1 more article gives this line's RRID in its Methods without naming the line nearby, so it is not listed.

PaperJournalYearHow we knowRetractedSource
PARP inhibition enhances the antitumor activity of HER3-DXd in non-small cell lung cancer.Cell Rep Med2026Its Methods give the RRID and name the linePubMed
Virtual Tumors Enable Prediction of Personalized Therapeutic Combinations for Non-Small Cell Lung Cancer.Cancer Res2026Its Methods give the RRID and name the linePubMed
Immune gene diversity and STING1 variants in shaping cancer immunity across different genetic ancestry populations.Cell Rep2026Its Methods give the RRID and name the linePubMed
Activity of Direct KRAS(G12C) Inhibitors in Preclinical Models of Pediatric Cancer.Mol Cancer Ther2026Its Methods give the RRID and name the linePubMed
Targeting CDK12/13 Drives Mitotic Arrest to Overcome Resistance to KRASG12C Inhibitors.Cancer Res2026Its Methods give the RRID and name the linePubMed
Wild-type RAS signaling is an essential therapeutic target in RAS-mutated cancers.Sci Signal2025Its Methods give the RRID and name the linePubMed
Glecirasib, a Potent and Selective Covalent KRAS G12C Inhibitor Exhibiting Synergism with Cetuximab or SHP2 Inhibitor JAB-3312.Cancer Res Commun2025Its Methods give the RRID and name the linePubMed
Impact of Co-mutations and Transcriptional Signatures in Non-Small Cell Lung Cancer Patients Treated with Adagrasib in the KRYSTAL-1 Trial.Clin Cancer Res2025Its Methods give the RRID and name the linePubMed
SOS1 Inhibition Enhances the Efficacy of KRASG12C Inhibitors and Delays Resistance in Lung Adenocarcinoma.Cancer Res2025Its Methods give the RRID and name the linePubMed
Targeting AKR1B10 by Drug Repurposing with Epalrestat Overcomes Chemoresistance in Non-Small Cell Lung Cancer Patient-Derived Tumor Organoids.Clin Cancer Res2024Its Methods give the RRID and name the linePubMed
In situ modeling of acquired resistance to RTK/RAS-pathway-targeted therapies.iScience2024Its Methods give the RRID and name the linePubMed
Discovery and Characterization of ZL-2201, a Potent, Highly Selective, and Orally Bioavailable Small-molecule DNA-PK Inhibitor.Cancer Res Commun2023Its Methods give the RRID and name the linePubMed
Systems-level analyses of protein-protein interaction network dysfunctions via epichaperomics identify cancer-specific mechanisms of stress adaptation.Nat Commun2023Its Methods give the RRID and name the linePubMed
TEAD Inhibitors Sensitize KRASG12C Inhibitors via Dual Cell Cycle Arrest in KRASG12C-Mutant NSCLC.Pharmaceuticals (Basel)2023Its Methods give the RRID and name the linePubMed
Sperm-specific COX6B2 enhances oxidative phosphorylation, proliferation, and survival in human lung adenocarcinoma.Elife2020Its Methods give the RRID and name the linePubMed
A Deregulated HOX Gene Axis Confers an Epigenetic Vulnerability in KRAS-Mutant Lung Cancers.Cancer Cell2020Its Methods give the RRID and name the linePubMed
RUVBL1/RUVBL2 ATPase Activity Drives PAQosome Maturation, DNA Replication and Radioresistance in Lung Cancer.Cell Chem Biol2020Its Methods give the RRID and name the linePubMed
From clinical specimens to human cancer preclinical models-a journey the NCI-cell line database-25 years later.J Cell Biochem2020Cellosaurus cites itPubMed
TR1801-ADC: a highly potent cMet antibody-drug conjugate with high activity in patient-derived xenograft models of solid tumors.Mol Oncol2020Its Methods give the RRID and name the linePubMed
Next-generation characterization of the Cancer Cell Line Encyclopedia.Nature2019Cellosaurus cites itPubMed

cell-lines-2026-11-14-0.1.0

Sources 4

  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
Each source's terms
  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

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