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Cell line · CVCL_1495

Papers51

PaperJournalYearHow we knowRetractedSource
Overcoming platinum resistance by the disulfiram metabolite CuET in novel preclinical models of human carcinomas.Mol Oncol2026Its Methods give the RRID and name the linePubMed
HES1 inhibition overcomes CDK4/6 inhibitor resistance by targeting cancer stemness in lung adenocarcinoma.J Exp Clin Cancer Res2026Its Methods give the RRID and name the linePubMed
Virtual Tumors Enable Prediction of Personalized Therapeutic Combinations for Non-Small Cell Lung Cancer.Cancer Res2026Its Methods give the RRID and name the linePubMed
Targeting KRAS Codon 13 Mutations Using Direct Combination Approaches in Non-Small Cell Lung Cancer.Cancer Discov2026Its Methods give the RRID and name the linePubMed
RASH3D19 mediates RAS activation through a positive feedback loop in KRAS-mutant cancer.Nat Cell Biol2026Its Methods give the RRID and name the linePubMed
RAS Mutation-Specific Responses to Paralog- and State-Selective RAS Inhibitors.Mol Cancer Res2026Its Methods give the RRID and name the linePubMed
Loss of UXS1 Selectively Depletes Pyrimidines and Induces Replication Stress in KEAP1-Mutant Lung Cancer.Cancer Res2025Its Methods give the RRID and name the linePubMed
Glecirasib, a Potent and Selective Covalent KRAS G12C Inhibitor Exhibiting Synergism with Cetuximab or SHP2 Inhibitor JAB-3312.Cancer Res Commun2025Its Methods give the RRID and name the linePubMed
Mutation of SMARCA4 Induces Cancer Cell-Intrinsic Defects in the Enhancer Landscape and Resistance to Immunotherapy.Cancer Res2025Its Methods give the RRID and name the linePubMed
The Selective WEE1 Inhibitor Azenosertib Shows Synergistic Antitumor Activity with KRASG12C Inhibitors in Preclinical Models.Cancer Res Commun2025Its Methods give the RRID and name the linePubMed
SOS1 Inhibition Enhances the Efficacy of KRASG12C Inhibitors and Delays Resistance in Lung Adenocarcinoma.Cancer Res2025Its Methods give the RRID and name the linePubMed
N-Myristoytransferase Inhibition Causes Mitochondrial Iron Overload and Parthanatos in TIM17A-Dependent Aggressive Lung Carcinoma.Cancer Res Commun2024Its Methods give the RRID and name the linePubMed
Targeting WEE1 enhances the antitumor effect of KRAS-mutated non-small cell lung cancer harboring TP53 mutations.Cell Rep Med2024Its Methods give the RRID and name the linePubMed
CRISPR-Cas9 Screening Identifies KRAS-Induced COX2 as a Driver of Immunotherapy Resistance in Lung Cancer.Cancer Res2024Its Methods give the RRID and name the linePubMed
Activation of polyamine catabolism promotes glutamine metabolism and creates a targetable vulnerability in lung cancer.Proc Natl Acad Sci U S A2024Its Methods give the RRID and name the linePubMed
FBXL16 promotes cell growth and drug resistance in lung adenocarcinomas with KRAS mutation by stabilizing IRS1 and upregulating IRS1/AKT signaling.Mol Oncol2024Its Methods give the RRID and name the linePubMed
MLK4 promotes glucose metabolism in lung adenocarcinoma through CREB-mediated activation of phosphoenolpyruvate carboxykinase and is regulated by KLF5.Oncogenesis2023Its Methods give the RRID and name the linePubMed
TEAD Inhibitors Sensitize KRASG12C Inhibitors via Dual Cell Cycle Arrest in KRASG12C-Mutant NSCLC.Pharmaceuticals (Basel)2023Its Methods give the RRID and name the linePubMed
Comprehensive Metabolic Tracing Reveals the Origin and Catabolism of Cysteine in Mammalian Tissues and Tumors.Cancer Res2023Its Methods give the RRID and name the linePubMed
Smoking-associated Downregulation of FILIP1L Enhances Lung Adenocarcinoma Progression Through Mucin Production, Inflammation, and Fibrosis.Cancer Res Commun2022Its Methods give the RRID and name the linePubMed

cell-lines-2026-11-14-0.1.0

Sources 4

  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
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  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

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