ConductScience.org, home

Cell line · CVCL_1724

Papers93

1 more article gives this line's RRID in its Methods without naming the line nearby, so it is not listed.

PaperJournalYearHow we knowRetractedSource
Targeting O6-methylguanine-DNA Methyltransferase Deficiency in Preclinical Models of Extracranial Human Cancers with a Tumor-Selective DNA-Modifying Agent.Mol Cancer Ther2026Its Methods give the RRID and name the linePubMed
CRISPR Screening Identifies SMARCAL1 and MRN as Modulators of WRN Dependency in MSI-H Colorectal Cancer.Cancer Res2026Its Methods give the RRID and name the linePubMed
Soluble Uric Acid Drives CD8+ T-cell Exhaustion by Inducing KSR1-Mediated MAPK Hyperactivation.Cancer Res2026Its Methods give the RRID and name the linePubMed
Radiation-induced MYB reduction unleashes TIM-3 expression in NK cells to attenuate antitumor immunity in colorectal cancer.J Transl Med2026Its Methods give the RRID and name the linePubMed
AURKA-mediated destabilization of SAPS3 drives ferroptosis evasion via 7-dehydrocholesterol biosynthesis in colorectal cancer.Cell Death Dis2026Its Methods give the RRID and name the linePubMed
Subcellular Redistribution of Endomembrane GPR15 Promotes NAD+-Mediated Metabolic Reprogramming and Boosts 5-FU Chemosensitivity in Colorectal Cancer.Cancer Res2026Its Methods give the RRID and name the linePubMed
Microsatellite Instable Cancer Cells Acquire On-target Resistance Mutations to WRN Helicase Inhibitors.Mol Cancer Ther2026Its Methods give the RRID and name the linePubMed
The dysadherin/carbonic anhydrase 9 axis shapes an acidic tumor microenvironment to promote colorectal cancer progression.Signal Transduct Target Ther2026Its Methods give the RRID and name the linePubMed
Cathepsin-D-mediated MHC class I degradation contributes to immune evasion in colorectal cancer.Cell Rep Med2026Its Methods give the RRID and name the linePubMed
Targeting WEE1 in ARID1A/TP53 Concurrent Mutant Colorectal Cancer by Exploiting R-Loop Accumulation and DNA Repair Deficiencies.Adv Sci (Weinh)2026Its Methods give the RRID and name the linePubMed
RAS Mutation-Specific Responses to Paralog- and State-Selective RAS Inhibitors.Mol Cancer Res2026Its Methods give the RRID and name the linePubMed
circ-EGFR is a predictor of response to Cetuximab and a potential target in colorectal cancer.EMBO Mol Med2025Its Methods give the RRID and name the linePubMed
Purinergic ecto-enzyme CD73 is a context-dependent tumor suppressor in colorectal cancer.J Biol Chem2025Its Methods give the RRID and name the linePubMed
GALNT7 Stratifies dMMR/MSI Colorectal Cancer into Distinct Molecular Subsets Associated with Prognosis and PD-L1 Expression.Cancer Res Commun2025Its Methods give the RRID and name the linePubMed
Pathway metabolite ratios reveal distinctive glutamine metabolism in a subset of proliferating cells.Mol Syst Biol2025Its Methods give the RRID and name the linePubMed
Clonal dispersal is associated with tumor heterogeneity and poor prognosis in colorectal cancer.iScience2025Its Methods give the RRID and name the linePubMed
Antibody-Drug Conjugates Targeting the EGFR Ligand Epiregulin Elicit Robust Antitumor Activity in Colorectal Cancer.Cancer Res2025Its Methods give the RRID and name the linePubMed
A Potent, Selective, Small-Molecule Inhibitor of DHX9 Abrogates Proliferation of Microsatellite Instable Cancers with Deficient Mismatch Repair.Cancer Res2025Its Methods give the RRID and name the linePubMed
Asparagine synthetase and G-protein coupled estrogen receptor are critical responders to nutrient supply in KRAS mutant colorectal cancer.Int J Cancer2025Its Methods give the RRID and name the linePubMed
The dysadherin/MMP9 axis modifies the extracellular matrix to accelerate colorectal cancer progression.Nat Commun2024Its Methods give the RRID and name the linePubMed

cell-lines-2026-11-14-0.1.0

Sources 4

  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
Each source's terms
  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

For software

Read this list from software.

Twenty rows a page, as JSON. No key.

GET https://conductscience.org/api/cell-lines/v1/CVCL_1724/papers