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Cell line · CVCL_1729

Papers62

PaperJournalYearHow we knowRetractedSource
Targeting CD24 Activates Macrophages to Reduce Tumor Burden in Preclinical Models of Solid Tumors.Clin Cancer Res2026Its Methods give the RRID and name the linePubMed
Beta-site Amyloid Precursor Protein Cleaving Enzyme 1 Overexpression Abrogates ST6GAL1 Mediated Rectal Cancer Chemoradiation Resistance.Cancer Genomics Proteomics2026Its Methods give the RRID and name the linePubMed
Tumor Invasive Border Index (TIBI) in colorectal cancer: linking infiltrative morphology to molecular insights.J Pathol2026Its Methods give the RRID and name the linePubMed
WEE1 kinase inhibition to overcome acquired resistance to targeted therapies in colorectal cancer.EMBO Mol Med2026Its Methods give the RRID and name the linePubMed
Constitutive EGFR Activation Induced by PTPRR Downregulation Confers Resistance to KRAS Inhibitors.Cancer Res Commun2026Its Methods give the RRID and name the linePubMed
USP10-mediated deubiquitination and activation of KRAS mutants promotes colorectal cancer via a novel USP10/KRAS positive feedback circuit.Acta Pharm Sin B2026Its Methods give the RRID and name the linePubMed
RASH3D19 mediates RAS activation through a positive feedback loop in KRAS-mutant cancer.Nat Cell Biol2026Its Methods give the RRID and name the linePubMed
Cytoplasmic p21 promotes stemness of colon cancer cells via activation of the NFκB pathway.Mol Oncol2026Its Methods give the RRID and name the linePubMed
Class IIa HDACs forced degradation allows resensitization of oxaliplatin-resistant FBXW7-mutated colorectal cancer.Mol Oncol2026Its Methods give the RRID and name the linePubMed
Therapeutic targeting of syndecan-1 axis overcomes acquired resistance to KRAS-targeted therapy in gastrointestinal cancers.Cell Rep Med2025Its Methods give the RRID and name the linePubMed
Glecirasib, a Potent and Selective Covalent KRAS G12C Inhibitor Exhibiting Synergism with Cetuximab or SHP2 Inhibitor JAB-3312.Cancer Res Commun2025Its Methods give the RRID and name the linePubMed
Direct Inhibition of RAS Reveals the Features of Oncogenic Signaling Driven by RAS G12 and Q61 Mutations.Cancer Discov2025Its Methods give the RRID and name the linePubMed
The Selective WEE1 Inhibitor Azenosertib Shows Synergistic Antitumor Activity with KRASG12C Inhibitors in Preclinical Models.Cancer Res Commun2025Its Methods give the RRID and name the linePubMed
Tumor Vessel Normalization via PFKFB3 Inhibition Alleviates Hypoxia and Increases Tumor Necrosis in Rectal Cancer upon Radiotherapy.Cancer Res Commun2024Its Methods give the RRID and name the linePubMed
AMT-562, a Novel HER3-targeting Antibody-Drug Conjugate, Demonstrates a Potential to Broaden Therapeutic Opportunities for HER3-expressing Tumors.Mol Cancer Ther2023Its Methods give the RRID and name the linePubMed
Alternative splicing of GSDMB modulates killer lymphocyte-triggered pyroptosis.Sci Immunol2023Its Methods give the RRID and name the linePubMed
The drug efflux pump MDR1 promotes intrinsic and acquired resistance to PROTACs in cancer cells.Sci Signal2022Its Methods give the RRID and name the linePubMed
Pan-cancer proteomic map of 949 human cell lines.Cancer Cell2022Cellosaurus cites itPubMed
Quantitative Proteomics of the Cancer Cell Line Encyclopedia.Cell2020Cellosaurus cites itPubMed
CoA Synthase (COASY) Mediates Radiation Resistance via PI3K Signaling in Rectal Cancer.Cancer Res2020Its Methods give the RRID and name the linePubMed

cell-lines-2026-11-14-0.1.0

Sources 4

  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
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  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

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