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Cell line · CVCL_2574

Papers30

PaperJournalYearHow we knowRetractedSource
High TMEM161A expression drives malignant phenotypes and predicts poor prognosis in colorectal cancer.iScience2026Its Methods give the RRID and name the linePubMed
WEE1 kinase inhibition to overcome acquired resistance to targeted therapies in colorectal cancer.EMBO Mol Med2026Its Methods give the RRID and name the linePubMed
BCL-xL as a therapeutic target in cetuximab-refractory colorectal cancer.Cell Death Dis2026Its Methods give the RRID and name the linePubMed
Cetuximab increases LGR5 expression and augments LGR5-targeting antibody-drug conjugate efficacy in patient-derived colorectal cancer models.Cell Rep Med2025Its Methods give the RRID and name the linePubMed
Cetuximab increases LGR5 expression and augments LGR5-targeting antibody-drug conjugate efficacy in patient-derived colorectal cancer models.bioRxiv2025Its Methods give the RRID and name the linePubMed
Direct Inhibition of RAS Reveals the Features of Oncogenic Signaling Driven by RAS G12 and Q61 Mutations.Cancer Discov2025Its Methods give the RRID and name the linePubMed
Antibody-Drug Conjugates Targeting the EGFR Ligand Epiregulin Elicit Robust Antitumor Activity in Colorectal Cancer.Cancer Res2025Its Methods give the RRID and name the linePubMed
PubMed 39605519bioRxiv2024Its Methods give the RRID and name the linePubMed
The MondoA-dependent TXNIP/GDF15 axis predicts oxaliplatin response in colorectal adenocarcinomas.EMBO Mol Med2024Its Methods give the RRID and name the linePubMed
ARAF protein kinase activates RAS by antagonizing its binding to RASGAP NF1.Mol Cell2022Its Methods give the RRID and name the linePubMed
Regulation of GTPase function by autophosphorylation.Mol Cell2022Its Methods give the RRID and name the linePubMed
Vulnerability to low-dose combination of irinotecan and niraparib in ATM-mutated colorectal cancer.J Exp Clin Cancer Res2021Its Methods give the RRID and name the linePubMed
Cetuximab-Mediated Protection from Hypoxia- Induced Cell Death: Implications for Therapy Sequence in Colorectal Cancer.Cancers (Basel)2020Its Methods give the RRID and name the linePubMed
An Interactive Resource to Probe Genetic Diversity and Estimated Ancestry in Cancer Cell Lines.Cancer Res2019Cellosaurus cites itPubMed
Genomic Determinants of Protein Abundance Variation in Colorectal Cancer Cells.Cell Rep2017Cellosaurus cites itPubMed
Highly Expressed Genes in Rapidly Proliferating Tumor Cells as New Targets for Colorectal Cancer Treatment.Clin Cancer Res2015Cellosaurus cites itPubMed
The molecular landscape of colorectal cancer cell lines unveils clinically actionable kinase targets.Nat Commun2015Cellosaurus cites itPubMed
Emergence of Multiple EGFR Extracellular Mutations during Cetuximab Treatment in Colorectal Cancer.Clin Cancer Res2015Cellosaurus cites itPubMed
Colorectal cancer cell lines are representative models of the main molecular subtypes of primary cancer.Cancer Res2014Cellosaurus cites itPubMed
Comparative N-glycan profiling of colorectal cancer cell lines reveals unique bisecting GlcNAc and α-2,3-linked sialic acid determinants are associated with membrane proteins of the more metastatic/aggressive cell lines.
J Proteome Res2014Cellosaurus cites itPubMed

cell-lines-2026-11-14-0.1.0

Sources 4

  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
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  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

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