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Cell line · CVCL_3422

Papers75

PaperJournalYearHow we knowRetractedSource
Targeting bromodomain and extra-terminal proteins reprograms macrophages and inhibits breast cancer progression.Front Immunol2026Its Methods give the RRID and name the linePubMed
In vivo CRISPR screening identifies metastasis suppressors in triple-negative breast cancer.Nat Commun2026Its Methods give the RRID and name the linePubMed
FET proteins and PARylation-dependent condensates promote replication fork reversal and genome stability.Nat Commun2026Its Methods give the RRID and name the linePubMed
Inhibition of cyclin-dependent kinases 12/13 using CT7439 as a treatment for colorectal cancer with CDK12 upregulation.Mol Oncol2026Its Methods give the RRID and name the linePubMed
Loss of IGF-1R impairs DNA-PKcs recruitment to chromatin leading to defective end-joining.Mol Oncol2026Its Methods give the RRID and name the linePubMed
An antibody-drug conjugate designed through clone and isotype selection restricts the growth of CSPG4-expressing triple-negative breast cancer.NPJ Precis Oncol2026Its Methods give the RRID and name the linePubMed
3D "Emboli" Culture Models Epithelial Breast Cancer Cell Oxidative Mitochondrial Metabolism with Relevance for Lung Metastasis.Cancer Res Commun2026Its Methods give the RRID and name the linePubMed
Camonsertib, an ATRi, in Combination with Low-Dose Gemcitabine in Solid Tumors with DNA Damage Response Aberrations: Preclinical and Phase Ib Results.Clin Cancer Res2026Its Methods give the RRID and name the linePubMed
Integrative CRISPR Screening and RNA Analyses Discover an Essential Role for PUF60 Interactions with 3' Splice Sites in Cancer Progression.Cancer Res2026Its Methods give the RRID and name the linePubMed
The CIP2A-TOPBP1 axis facilitates mitotic DNA repair via MiDAS and MMEJ.Nat Commun2025Its Methods give the RRID and name the linePubMed
MYC inhibition by Omomyc causes DNA damage and overcomes PARPi resistance in breast cancer.Cell Rep2025Its Methods give the RRID and name the linePubMed
High-throughput combination screening of Pidnarulex and other G-quadruplex ligands in multi-cell type tumor spheroids.SLAS Discov2025Its Methods give the RRID and name the linePubMed
PARP inhibitor and PRMT5 inhibitor synergy is independent of BRCA1/2 and MTAP status in breast cancer cells.Sci Rep2025Its Methods give the RRID and name the linePubMed
PCBP2 Mediates Olaparib Resistance in Breast Cancer by Inhibiting m6A Methylation to Stabilize PARP1 mRNA.Cancer Res2025Its Methods give the RRID and name the linePubMed
Curcumin Induces Homologous Recombination Deficiency by BRCA2 Degradation in Breast Cancer and Normal Cells.Cancers (Basel)2025Its Methods give the RRID and name the linePubMed
Depleting the Action of EZH2 through PI3K-mTOR Inhibition to Overcome Metastasis and Immunotherapy Resistance in Triple-Negative Breast Cancer.Mol Cancer Ther2025Its Methods give the RRID and name the linePubMed
E-Cadherin Is a Structuring Component of Invadopodia in Pancreatic Cancer.J Cell Mol Med2025Its Methods give the RRID and name the linePubMed
Inhibition of MCL-1 and MEK Overcomes MEK Inhibitor Resistance in Triple-Negative and Inflammatory Breast Cancers.Mol Cancer Ther2025Its Methods give the RRID and name the linePubMed
Defining the regulatory logic of breast cancer using single-cell epigenetic and transcriptome profiling.Cell Genom2025Its Methods give the RRID and name the linePubMed
Inhibition of proteolytic and ATPase activities of the proteasome by the BTK inhibitor CGI-1746.iScience2024Its Methods give the RRID and name the linePubMed

cell-lines-2026-11-14-0.1.0

Sources 4

  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
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  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

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