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Cell line · CVCL_5160

Papers34

PaperJournalYearHow we knowRetractedSource
Systematic Targeting of Protein Complexes with Molecular COUPLrs.Cancer Discov2026Its Methods give the RRID and name the linePubMed
Integration and validation of complementary ex vivo assays for functional precision oncology.NPJ Precis Oncol2026Its Methods give the RRID and name the linePubMed
FET Fusion Oncoproteins Disrupt Physiologic DNA Repair and Create a Targetable Opportunity for ATR Inhibitor Therapy.Cancer Res2026Its Methods give the RRID and name the linePubMed
A comprehensive functional atlas of ALK kinase domain variants reveals resistance landscape to ALK inhibitors.Genome Biol2026Its Methods give the RRID and name the linePubMed
Clinical Characteristics and Spatial Transcriptome Analysis of Non-Small Cell Lung Cancers Exhibiting Early Alectinib Resistance: A Retrospective OLCSG Study.Cancer Res Commun2026Its Methods give the RRID and name the linePubMed
Cancer-associated fibroblasts promote drug resistance in ALK-driven lung adenocarcinoma cells by upregulating lipid biosynthesis.Cancer Metab2025Its Methods give the RRID and name the linePubMed
Amplified dosage of the NKX2-1 lineage transcription factor controls its oncogenic role in lung adenocarcinoma.Mol Cell2025Its Methods give the RRID and name the linePubMed
Integrative transcriptomic analysis identifies emetine as a promising candidate for overcoming acquired resistance to ALK inhibitors in lung cancer.Mol Oncol2025Its Methods give the RRID and name the linePubMed
AXL and SHC1 confer crizotinib resistance in patient-derived xenograft model of ALK-driven lung cancer.iScience2024Its Methods give the RRID and name the linePubMed
PubMed 37994369bioRxiv2023Its Methods give the RRID and name the linePubMed
ALK peptide vaccination restores the immunogenicity of ALK-rearranged non-small cell lung cancer.Nat Cancer2023Its Methods give the RRID and name the linePubMed
Systems-level analyses of protein-protein interaction network dysfunctions via epichaperomics identify cancer-specific mechanisms of stress adaptation.Nat Commun2023Its Methods give the RRID and name the linePubMed
PI3Kβ inhibition enhances ALK-inhibitor sensitivity in ALK-rearranged lung cancer.Mol Oncol2023Its Methods give the RRID and name the linePubMed
Pan-cancer proteomic map of 949 human cell lines.Cancer Cell2022Cellosaurus cites itPubMed
Loss of spindly sensitizes EML4-ALK v3 lung cancer cells to HSP90 inhibitors.2022Cellosaurus cites itDOI
A MET-PTPRK kinase-phosphatase rheostat controls ZNRF3 and Wnt signaling.Elife2021Its Methods give the RRID and name the linePubMed
Silibinin Suppresses Tumor Cell-Intrinsic Resistance to Nintedanib and Enhances Its Clinical Activity in Lung Cancer.Cancers (Basel)2021Its Methods give the RRID and name the linePubMed
RUVBL1/RUVBL2 ATPase Activity Drives PAQosome Maturation, DNA Replication and Radioresistance in Lung Cancer.Cell Chem Biol2020Its Methods give the RRID and name the linePubMed
From clinical specimens to human cancer preclinical models-a journey the NCI-cell line database-25 years later.J Cell Biochem2020Cellosaurus cites itPubMed
Radioresistance of KRAS/TP53-mutated lung cancer can be overcome by radiation dose escalation or EGFR tyrosine kinase inhibition in vivo.Int J Cancer2020Its Methods give the RRID and name the linePubMed

cell-lines-2026-11-14-0.1.0

Sources 4

  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
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  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

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