ConductScience.org, home

Cell line · CVCL_6027

Papers32

PaperJournalYearHow we knowRetractedSource
Hexokinase2-engineered T cells display increased anti-tumor function.Front Immunol2025Its Methods give the RRID and name the linePubMed
Targeting TGFβ with chimeric switch receptor and secreted trap to improve T cells anti-tumor activity.Front Immunol2024Its Methods give the RRID and name the linePubMed
Genetically engineering glycolysis in T cells increases their antitumor function.J Immunother Cancer2024Its Methods give the RRID and name the linePubMed
The CD73 immune checkpoint promotes tumor cell metabolic fitness.Elife2023Its Methods give the RRID and name the linePubMed
An adverse tumor-protective effect of IDO1 inhibition.Cell Rep Med2023Its Methods give the RRID and name the linePubMed
A TIGIT-based chimeric co-stimulatory switch receptor improves T-cell anti-tumor function.J Immunother Cancer2019Its Methods give the RRID and name the linePubMed
TCLP: an online cancer cell line catalogue integrating HLA type, predicted neo-epitopes, virus and gene expression.Genome Med2015Cellosaurus cites itPubMed
A resource for cell line authentication, annotation and quality control.Nature2015Cellosaurus cites itPubMed
A comprehensive transcriptional portrait of human cancer cell lines.Nat Biotechnol2015Cellosaurus cites itPubMed
Loss of NF1 in cutaneous melanoma is associated with RAS activation and MEK dependence.Cancer Res2014Cellosaurus cites itPubMed
Investigating the role of melanin in UVA/UVB- and hydrogen peroxide-induced cellular and mitochondrial ROS production and mitochondrial DNA damage in human melanoma cells.Free Radic Biol Med2012Cellosaurus cites itPubMed
Concurrent loss of the PTEN and RB1 tumor suppressors attenuates RAF dependence in melanomas harboring (V600E)BRAF.Oncogene2012Cellosaurus cites itPubMed
Lack of extracellular signal-regulated kinase mitogen-activated protein kinase signaling shows a new type of melanoma.Cancer Res2007Cellosaurus cites itPubMed
Involvement of overexpressed wild-type BRAF in the growth of malignant melanoma cell lines.Oncogene2004Cellosaurus cites itPubMed
Genetic interaction between NRAS and BRAF mutations and PTEN/MMAC1 inactivation in melanoma.J Invest Dermatol2004Cellosaurus cites itPubMed
Relative reciprocity of NRAS and PTEN/MMAC1 alterations in cutaneous melanoma cell lines.Cancer Res2000Cellosaurus cites itPubMed
Identification of PTEN/MMAC1 alterations in uncultured melanomas and melanoma cell lines.Oncogene1998Cellosaurus cites itPubMed
Stringent allele/epitope requirements for MART-1/Melan A immunodominance: implications for peptide-based immunotherapy.J Immunol1998Cellosaurus cites itPubMed
Characterization of gene regulatory elements for selective gene expression in human melanoma cells.Biochem Biophys Res Commun1995Cellosaurus cites itPubMed
Production and characterization of antibodies against human tyrosinase.J Invest Dermatol1994Cellosaurus cites itPubMed

cell-lines-2026-11-14-0.1.0

Sources 4

  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
Each source's terms
  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

For software

Read this list from software.

Twenty rows a page, as JSON. No key.

GET https://conductscience.org/api/cell-lines/v1/CVCL_6027/papers