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Cell line · CVCL_7916

Papers26

PaperJournalYearHow we knowRetractedSource
Nicotinic Acid Restriction Potentiates NAMPT Inhibition in Acute Myeloid Leukemia Xenograft Models.Cancer Res Commun2026Its Methods give the RRID and name the linePubMed
Development of CAR T Cells Targeting a Surface RNA-Binding Protein for the Treatment of Acute Leukemias.Cancer Discov2026Its Methods give the RRID and name the linePubMed
Enhancement of CD117-Targeted Bispecific T-cell Engagement by CD33-Targeted Bispecific T-cell Costimulation in Acute Myeloid Leukemia.Cancer Res Commun2026Its Methods give the RRID and name the linePubMed
A Bispecific Anti-Fluorescein × Anti-CD3 T-cell Engager in Combination with Fluoresceinated Adaptors Enables Lysis of AML Cells.Mol Cancer Ther2026Its Methods give the RRID and name the linePubMed
Activity of Direct KRAS(G12C) Inhibitors in Preclinical Models of Pediatric Cancer.Mol Cancer Ther2026Its Methods give the RRID and name the linePubMed
Nutrient Availability Dictates Cancer Metabolism-Based Therapeutic Responses to Nononcology Drugs.Cancer Res2026Its Methods give the RRID and name the linePubMed
An Isoform-Specific RUNX1C-BTG2 Axis Governs AML Quiescence and Chemoresistance.Blood Cancer Discov2025Its Methods give the RRID and name the linePubMed
Catalytic Inhibition of KAT6/KAT7 Enhances the Efficacy and Overcomes Primary and Acquired Resistance to Menin Inhibitors in MLL Leukemia.Cancer Discov2025Its Methods give the RRID and name the linePubMed
BKT300: A Novel Anti-Leukemic Small Molecule Targeting the Protein Regulator of Cytokinesis 1 (PRC1) PathwayRes Sq2025Its Methods give the RRID and name the linePubMed
Targeting rapid TKI-induced AXL upregulation overcomes adaptive ERK reactivation and exerts antileukemic effects in FLT3/ITD acute myeloid leukemia.Mol Oncol2025Its Methods give the RRID and name the linePubMed
The AXL inhibitor bemcentinib overcomes microenvironment-mediated resistance to pioglitazone in acute myeloid leukemia.FEBS J2025Its Methods give the RRID and name the linePubMed
Pharmacological inhibition of RAS overcomes FLT3 inhibitor resistance in FLT3-ITD+ AML through AP-1 and RUNX1.iScience2024Its Methods give the RRID and name the linePubMed
Pim Kinase Inhibitors Increase Gilteritinib Cytotoxicity in FLT3-ITD Acute Myeloid Leukemia Through GSK-3β Activation and c-Myc and Mcl-1 Proteasomal Degradation.Cancer Res Commun2024Its Methods give the RRID and name the linePubMed
Allosteric SHP2 inhibition increases apoptotic dependency on BCL2 and synergizes with venetoclax in FLT3- and KIT-mutant AML.Cell Rep Med2023Its Methods give the RRID and name the linePubMed
PARP-1 improves leukemia outcomes by inducing parthanatos during chemotherapy.Cell Rep Med2023Its Methods give the RRID and name the linePubMed
Targeted kinase degradation via the KLHDC2 ubiquitin E3 ligase.Cell Chem Biol2023Its Methods give the RRID and name the linePubMed
Use of Payload Binding Selectivity Enhancers to Improve Therapeutic Index of Maytansinoid-Antibody-Drug Conjugates.Mol Cancer Ther2023Its Methods give the RRID and name the linePubMed
Targeting MET and FGFR in Relapsed or Refractory Acute Myeloid Leukemia: Preclinical and Clinical Findings, and Signal Transduction Correlates.Clin Cancer Res2023Its Methods give the RRID and name the linePubMed
Subversion of Serotonin Receptor Signaling in Osteoblasts by Kynurenine Drives Acute Myeloid Leukemia.Cancer Discov2022Its Methods give the RRID and name the linePubMed
A method for overcoming plasma protein inhibition of tyrosine kinase inhibitors.Blood Cancer Discov2021Its Methods give the RRID and name the linePubMed

cell-lines-2026-11-14-0.1.0

Sources 4

  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
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  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

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