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Cell line · CVCL_8794

Papers18

PaperJournalYearHow we knowRetractedSource
Development of humanized mouse models for the preclinical evaluation of NK cell-based therapies.Front Immunol2026Its Methods give the RRID and name the linePubMed
CD47 promotes myeloma cell growth through JAK-STAT3/MYC signalling and is targetable by ruxolitinib.Br J Haematol2026Its Methods give the RRID and name the linePubMed
Targeting BCMA in multiple myeloma with a trifunctional NK cell engager.Cell Rep Med2026Its Methods give the RRID and name the linePubMed
Self-Assembling Multi-Antigen T Cell Hybridizers for Precision Immunotherapy of Multiple Myeloma.Adv Healthc Mater2025Its Methods give the RRID and name the linePubMed
Inhibition of acyl-CoA synthetase long-chain isozymes decreases multiple myeloma cell proliferation and causes mitochondrial dysfunction.Mol Oncol2025Its Methods give the RRID and name the linePubMed
Kinetics of Nirogacestat-Mediated Increases in B-cell Maturation Antigen on Plasma Cells Inform Therapeutic Combinations in Multiple Myeloma.Cancer Res Commun2024Its Methods give the RRID and name the linePubMed
Preclinical Evaluation of STI-8811, a Novel Antibody-Drug Conjugate Targeting BCMA for the Treatment of Multiple Myeloma.Cancer Res Commun2024Its Methods give the RRID and name the linePubMed
Multiple mechanisms contribute to acquired TRAIL resistance in multiple myeloma.Cancer Cell Int2024Its Methods give the RRID and name the linePubMed
CRIP1 involves the pathogenesis of multiple myeloma via dual-regulation of proteasome and autophagy.EBioMedicine2024Its Methods give the RRID and name the linePubMed
Systems-level analyses of protein-protein interaction network dysfunctions via epichaperomics identify cancer-specific mechanisms of stress adaptation.Nat Commun2023Its Methods give the RRID and name the linePubMed
miR-17-3p promotes the proliferation of multiple myeloma cells by downregulating P21 expression through LMLN inhibition.Int J Cancer2021Its Methods give the RRID and name the linePubMed
Oxidatively modified low-density lipoproteins are potential mediators of proteasome inhibitor resistance in multiple myeloma.Int J Cancer2021Its Methods give the RRID and name the linePubMed
Physical and Functional Analysis of the Putative Rpn13 Inhibitor RA190.Cell Chem Biol2020Its Methods give the RRID and name the linePubMed
Evaluating the efficacy of multiple myeloma cell lines as models for patient tumors via transcriptomic correlation analysis.Leukemia2020Cellosaurus cites itPubMed
Characterization of Human Cancer Cell Lines by Reverse-phase Protein Arrays.Cancer Cell2017Cellosaurus cites itPubMed
Targeted capture and sequencing of immunoglobulin rearrangements in multiple myeloma to enable detection of minimal residual disease.2017Cellosaurus cites itCellosaurus
Promiscuous mutations activate the noncanonical NF-kappaB pathway in multiple myeloma.Cancer Cell2007Cellosaurus cites itPubMed
Characterization of the MM.1 human multiple myeloma (MM) cell lines: a model system to elucidate the characteristics, behavior, and signaling of steroid-sensitive and -resistant MM cells.Exp Hematol2003Cellosaurus cites itPubMed

cell-lines-2026-11-14-0.1.0

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  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
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  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

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