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Cell line · CVCL_9723

Papers12

PaperJournalYearHow we knowRetractedSource
Resistance to membrane damage enhances metastatic potential of breast cancer cells via dysregulated phospholipid profiles and annexin-mediated repair.Cell Mol Life Sci2026Its Methods give the RRID and name the linePubMed
Targeting Cystine Addiction Suppresses Breast Cancer Lung Metastasis.Cancer Res2026Its Methods give the RRID and name the linePubMed
ICAM1high Neutrophils Sculpt Tumor Evolution and Metastasis Through Symbiotic Adhesion and Reverse Migration.Cancer Res2026Its Methods give the RRID and name the linePubMed
Whole-genome doubling drives immune evasion by silencing antigen presentation.Cancer Cell2026Its Methods give the RRID and name the linePubMed
Distinct Endothelial Phenotype Associates with Macrophage-Enriched Microenvironments in Triple-Negative Breast Cancer.Res Sq2026Its Methods give the RRID and name the linePubMed
Peripheral blood proteome biomarkers distinguish immunosuppressive features of cancer progression.Mol Oncol2025Its Methods give the RRID and name the linePubMed
The role of microglia in 67NR mammary tumor-induced suppression of brain responses to immune challenges in female mice.J Neurochem2024Its Methods give the RRID and name the linePubMed
ZBTB18 restricts chromatin accessibility and prevents transcriptional adaptations that drive metastasis.Sci Adv2023Its Methods give the RRID and name the linePubMed
Low lamin A levels enhance confined cell migration and metastatic capacity in breast cancer.Oncogene2022Its Methods give the RRID and name the linePubMed
Genomic analysis of a spontaneous model of breast cancer metastasis to bone reveals a role for the extracellular matrix.Mol Cancer Res2005Cellosaurus cites itPubMed
A novel orthotopic model of breast cancer metastasis to bone.Clin Exp Metastasis1999Cellosaurus cites itPubMed
Selective events in the metastatic process defined by analysis of the sequential dissemination of subpopulations of a mouse mammary tumor.Cancer Res1992Cellosaurus cites itPubMed

cell-lines-2026-11-14-0.1.0

Sources 4

  • CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
  • PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
  • NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
  • PubMedNLM's terms
Each source's terms
  • CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
  • PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
  • NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
  • PubMedNLM's terms

Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.

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