Cell line · CVCL_EQ22
GES-1
Transformed cell line, Homo sapiens. 46 papers.
4 sourcescell-lines-2026-11-14-0.1.0
Cite this record
ConductScience Library. GES-1. Cell lines, record CVCL_EQ22. Release cell-lines-2026-11-14-0.1.0, 14 Nov 2026. https://conductscience.org/library/cell-lines/CVCL_EQ22
On this page
Cell line
As Cellosaurus gives it.
- Name
- GES-1
- Accession
- CVCL_EQ22
- RRID
- RRID:CVCL_EQ22
- Synonyms
- GES1
- Species
- Homo sapiens (Human)
- Sex
- Sex unspecified
- Age at sampling
- 9FM
- Category
- Transformed cell line
- Derived from
- Fetal stomach, epithelium (in situ)
- Cell type
- Epithelial cell of stomach
- Entry created
- 2016-09-26
- Entry updated
- 2025-04-10
Source Cellosaurus
Problems0
Cellosaurus records no problem for this cell line.
None in this release.
Papers46
2 more articles give this line's RRID in its Methods without naming the line nearby, so they are not listed.
| Paper | Journal | Year | How we know | Retracted | Source |
|---|---|---|---|---|---|
| EIF3I reprograms iron metabolism to promote gastric cancer progression by stabilizing YAP. | Cell Mol Biol Lett | 2026 | Its Methods give the RRID and name the line | PubMed | |
| Epigenetic Activation of ANGPTL4 Drives EBV-Negative Gastric Cancer via Neutrophil Recruitment and NET Release via Stabilizing ANXA5. | Adv Sci (Weinh) | 2026 | Its Methods give the RRID and name the line | PubMed | |
| Lactobacillus salivarius potentiates gastrointestinal cancer immunotherapy through its metabolite chenodeoxycholic acid. | Cell Rep Med | 2026 | Its Methods give the RRID and name the line | PubMed | |
| METTL3-Mediated m6A Modification of COL5A2 Inhibits Ferroptosis Through an IGF2BP3-dependent Mechanism in Gastric Cancer. | Technol Cancer Res Treat | 2026 | Its Methods give the RRID and name the line | PubMed | |
| Luteolin as a urease inhibitor: A novel therapeutic strategy against Helicobacter pylori. | Virulence | 2026 | Its Methods give the RRID and name the line | PubMed | |
| METTL7A Downregulation Drives SLC1A5-Mediated Glutamine Competition to Promote Tumor Proliferation and Suppress CD8+ T Cell Immunity in Gastric Cancer. | Adv Sci (Weinh) | 2026 | Its Methods give the RRID and name the line | PubMed | |
| Disrupting Helicobacter pylori Iron Homeostasis With Bismuth Nanodrug-Mediated Nutritional Trap for Targeted Gastric Infection Therapy. | Adv Sci (Weinh) | 2026 | Its Methods give the RRID and name the line | PubMed | |
| Long non-coding RNA SOCS2-AS1 inhibits gastric cancer progression via the miR-324-3p/GRIK3 axis: a mechanistic study. | World J Surg Oncol | 2026 | Its Methods give the RRID and name the line | PubMed | |
| Inhibition of 5'-tiRNAGly restores anti-PD-1 immunotherapy efficacy via DLST-mediated OGDHL succinylation in gastric cancer. | J Exp Clin Cancer Res | 2026 | Its Methods give the RRID and name the line | PubMed | |
| The Role of NADPH Oxidase 4 in Neutrophil-Mediated Immune Escape in Gastric Cancer. | Int J Biol Sci | 2026 | Its Methods give the RRID and name the line | PubMed |
1 to 10 of 46
See all 46 papersRelatives11
| Relation | Cell line | Accession | Problem | Source |
|---|---|---|---|---|
| Derived from it | GES-1/miR-23a | CVCL_VL75 | Cell lines | |
| Derived from it | HyCyte GES-1 KO-hCD14 | CVCL_E1JV | Cell lines | |
| Derived from it | HyCyte GES-1 KO-hCTSB | CVCL_F1NT | Cell lines | |
| Derived from it | HyCyte GES-1 KO-hDDX17 | CVCL_F1NU | Cell lines | |
| Derived from it | HyCyte GES-1 KO-hFLVCR1 | CVCL_F4FF | Cell lines | |
| Derived from it | HyCyte GES-1 KO-hGBP5 | CVCL_F1NV | Cell lines | |
| Derived from it | HyCyte GES-1 KO-hGSDMD | CVCL_D6B6 | Cell lines | |
| Derived from it | HyCyte GES-1 KO-hIGF1R | CVCL_F4FC | Cell lines | |
| Derived from it | HyCyte GES-1 KO-hPGAM5 | CVCL_F4JF | Cell lines | |
| Derived from it | HyCyte GES-1 KO-hTRIM47 | CVCL_F4GZ | Cell lines |
1 to 10 of 11
See all 11 relativesCell banks1
| Cell bank | Catalog number | Status |
|---|---|---|
| CLS | 305428 |
Source CLS
Data dates
The date of each source this release read.
Sources 4
- CellosaurusCC BY 4.0, with credit to Cellosaurus (SIB)
- PubMed Central's articlesTitles shown only for CC0, CC BY and public domain articles
- NLM's PMC Article DatasetsCourtesy of NLM, which does not endorse this database
- PubMedNLM's terms
Each source's terms
- CellosaurusCC BY 4.0. Cellosaurus's README: "we have chosen to apply the Creative Commons Attribution 4.0 International (CC BY 4.0) license to Cellosaurus (https://creativecommons.org/licenses/by/4.0/). This means that you are free to copy and redistribute Cellosaurus in any medium or format. You can remix, transform, and build upon Cellosaurus for any purpose, even commercially. You must give appropriate credit, provide a link to the license, and indicate if changes were made." Copyright (c) CALIPHO group, SIB Swiss Institute of Bioinformatics. Read on 14 Nov 2026 at https://ftp.expasy.org/databases/cellosaurus/README
- PubMed Central's articlesEach article's own license. Its words show only under CC0, CC BY or public domain
- NLM's PMC Article DatasetsNLM's PMC Article Datasets on AWS, https://registry.opendata.aws/ncbi-pmc. Its terms: users "acknowledge NLM as the source of the data in a clear and conspicuous manner", do not use the PubMed Central wordmark or the PMC logo, do not "indicate or imply that NLM/NIH/HHS has endorsed" a product, and "only distribute data that are licensed for redistribution". Each article keeps its own license: https://pmc.ncbi.nlm.nih.gov/about/copyright/
- PubMedNLM's terms
Cellosaurus, SIB Swiss Institute of Bioinformatics, https://www.cellosaurus.org, CC BY 4.0. Its problem notes are given in its own words.
For software
Read this cell line from software.
The record and each of its lists are JSON, twenty rows a page. No key.
GET https://conductscience.org/api/cell-lines/v1/CVCL_EQ22
GET https://conductscience.org/api/cell-lines/v1/CVCL_EQ22/papers?page=2