ConductScience.org, home
NKRF: NFKB repressing factor
API referenceFor AI assistants

Gene · 55922

Papers1,288

Newest first. Swiss-Prot's references are curated; PubTator's text mining finds papers that name the gene, not always ones about it.

1 to 20 of 1,288

Download this page as JSON
TitleJournalYearPMIDFound bySource
CHST1 Drives Immunotherapy Resistance in Triple-Negative Breast Cancer by Orchestrating an Immunosuppressive Microenvironment via the NKRF-CCL20-Macrophage Axis.Adv Sci (Weinh)202642635979PubTator, text miningPubMed
The Reproductive Toxicity of Microplastics: From Environmental Contamination to Organ Distribution and Reproductive Hazards.Environ Toxicol202642528124PubTator, text miningPubMed
Proteolytic activation of c-MYC facilitated by DOT1L.Proc Natl Acad Sci U S A202642520114PubTator, text miningPubMed
Hydroxylamine steers nitrogen metabolism toward dissimilatory nitrate reduction to ammonium by suppressing competitive denitrification.Bioresour Technol202642398553PubTator, text miningPubMed
Ribonuclease DIS3 delays aging and senescence by generating tRNA halves.Nat Commun202642168172PubTator, text miningPubMed
Cathepsin B protease mediates high population density-induced mutagenesis to drive genome evolution and competitive growth.Nat Commun202642082483PubTator, text miningPubMed
TRIM26-mediated NKRF degradation drives Osimertinib resistance through SNRPD2-dependent stress granule formation in lung adenocarcinoma.Cell Death Dis202642026030PubTator, text miningPubMed
Repurposing Hetrombopag for Multiple Myeloma by Targeting PNPO: A Celastrol-Inspired Approach.Basic Clin Pharmacol Toxicol202641914238PubTator, text miningPubMed
Crosstalk between KRAS and miRNAs in pancreatic cancer: Opportunities for its diagnosis, prognosis and therapeutic intervention (Review).Int J Oncol202641860028PubTator, text miningPubMed
Biochar enhances nitrogen removal capacity in sulfur-driven autotrophic denitrification at low temperatures: Performance evaluation, microbial structure analysis, and metabolic pathway reconstruction.Bioresour Technol202641856334PubTator, text miningPubMed
Ferroptosis in cardiovascular diseases: molecular mechanisms and a novel therapeutic target.Mol Biomed202641795043PubTator, text miningPubMed
Mitochondrial function meets oncology: the multifaceted role of TFAM across cancer types.Apoptosis202641774266PubTator, text miningPubMed
Hepatic mitochondrial signaling as a systemic hub: inter-organ communication networks in aging and aging-related diseases.Front Cell Dev Biol202641768994PubTator, text miningPubMed
Global research trends and hotspots in mesenchymal stem cell applications for bone regeneration: A bibliometric and visualized analysis.Medicine (Baltimore)202641760007PubTator, text miningPubMed
Simufilam in Alzheimer's Disease: Assessment of Efficacy of a Controversial Drug in Human Neuronal Cell Culture.Pharmaceuticals (Basel)202641754821PubTator, text miningPubMed
Nuclear Receptor-Targeted Therapies: Reprogramming Metabolism with TRβ, ERRα, and LXR Modulators.Biomolecules202641750342PubTator, text miningPubMed
Higher Purity of Phosphatidylserine Improves Human Cortical Neuron Function by Modulating SIRT1-PGC-1α Pathways.Brain Sci202641750195PubTator, text miningPubMed
The Cross-Knit Between Immune Cells and Thyroid Function in Autoimmune Thyroid Disorders: What We Can Learn from Inborn Errors of Immunity.Children (Basel)202641749525PubTator, text miningPubMed
Mitochondria at the Crossroads of Cardiovascular Disease: Mechanistic Drivers and Emerging Therapeutic Strategies.Cells202641744815PubTator, text miningPubMed
Partially shared multi-modal embedding learns holistic representation of cell state.Nat Comput Sci202641741805PubTator, text miningPubMed

gene-search-2026-11-11-0.2.0

Next page

Sources 12

  • ClinicalTrials.gov's posted results, through CTTI's AACTFreely available. Acknowledgement requested
  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • EMBL-EBI's patent proteinsNo added restrictions. Attribution expected
  • GenBank's patent filesNo restrictions from NCBI. Submitters may hold rights
  • HGNCCC0
  • NCBI GenePublic domain (US government)
  • NIH's project files and grant-paper linksPublic domain
  • Open TargetsCC0 1.0
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ClinicalTrials.gov's posted results, through CTTI's AACTClinicalTrials.gov data via Aggregate Analysis of ClinicalTrials.gov (AACT), CTTI. CTTI makes its materials freely available and requests acknowledgement. https://aact.ctti-clinicaltrials.org/documentation/299
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • EMBL-EBI's patent proteinsEMBL-EBI's patent protein files from the EPO, JPO, KIPO and USPTO. EMBL-EBI: "EMBL-EBI itself places no additional restrictions on the use or redistribution of the data available via its Data Resources and Tools other than those provided by the original data owners" and "EMBL-EBI expects attribution (e.g. in publications, services or products) for the use of any of its Data Resources and Tools." Read on 11 Nov 2026 at https://www.ebi.ac.uk/about/terms-of-use
  • GenBank's patent filesGenBank's patent files. NCBI: "NCBI places no restrictions on the use or distribution of the GenBank data. However, some submitters may claim patent, copyright, or other intellectual property rights in all or a portion of the data they have submitted." Read on 11 Nov 2026 at https://www.ncbi.nlm.nih.gov/genbank/about/
  • HGNCCC0: https://www.genenames.org/about/license/
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH's project files and grant-paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

For software

Read this list from software.

Twenty rows a page, as JSON. No key.

GET https://conductscience.org/api/genes/v1/55922/papers

API referenceFor AI assistants