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Paper10692040

Recessive expression of the H2A-controlled immune response phenotype depends critically on antigen dose.

Barcenas-Morales G and 4 others. Immunology, 2000. Cited by 3 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Recessive expression of the H2A-controlled immune response phenotype depends critically on antigen dose. Papers, record 10692040. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/10692040

Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Recessive expression of the H2A-controlled immune response phenotype depends critically on antigen dose.
PubMed
Journal
Immunology
PubMed
Year
2000
PubMed
Publication types
Journal Article
PubMed
Citations, by iCite
3
iCite
Relative citation ratio, by iCite
0.08
iCite
Revised in PubMed
2019-05-13
PubMed

Authors5

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Barcenas-Morales GG
2Merkenschlager MM
3Wahid FF
4Döffinger RR
5Ivanyi JJ

Source PubMed

Topics16

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Antigens, BacterialMajor topic
HistonesMajor topic
Mycobacterium tuberculosisMajor topic
Alleles
Animals
Antibodies, Bacterial
Antigen Presentation
Dose-Response Relationship, Immunologic
Female
Gene Dosage
Haplotypes
Mice
Mice, Inbred C57BL
Mice, Inbred Strains
Peptide Fragments
T-Lymphocytes

Source PubMed

References36

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
11,194,855Genetic control of specific immune suppression. II. H-2-linked dominant genetic control of immune suppression by the random copolymer L-glutamic acid50-L-tyrosine50 (GT).J Exp Med1975https://conductscience.org/library/papers/1194855PubMed
21,194,856Dominant nonresponsiveness in the induction of autoimmunity to liver-specific F antigen.J Exp Med1975https://conductscience.org/library/papers/1194856PubMed
31,279,442Signalling through the MHC class II cytoplasmic domain is required for antigen presentation and induces B7 expression.Nature1992https://conductscience.org/library/papers/1279442PubMed
41,672,336Dominant low responsiveness in the IgG response of mice to the complex protein antigen type 1 fimbriae from Actinomyces viscosus T14V.J Immunol1991https://conductscience.org/library/papers/1672336PubMed
51,909,605Mice lacking MHC class II molecules.Cell1991https://conductscience.org/library/papers/1909605PubMed
61,970,351Co-dominant restriction by a mixed-haplotype I-A molecule (alpha k beta b) for the lysozyme peptide 52-61 in H-2k x H-2b F1 mice.J Immunol1990https://conductscience.org/library/papers/1970351PubMed
71,975,796Dominant reduced responsiveness controlled by H-2(Kb)Ab. A new pattern evoked by Thy-1 antigen and F liver antigen.Immunogenetics1990https://conductscience.org/library/papers/1975796PubMed
82,414,111Insulin-specific T cell hybridomas derived from (H-2b x H-2k)F1 mice preferably employ F1-unique restriction elements for antigen recognition.Eur J Immunol1985https://conductscience.org/library/papers/2414111PubMed
92,431,994Control by H-2 genes of murine antibody responses to protein antigens of Mycobacterium tuberculosis.Immunology1986https://conductscience.org/library/papers/2431994PubMed
102,467,772Immune suppression genes.Clin Exp Immunol1989https://conductscience.org/library/papers/2467772PubMed
1125,291,681I-E expression and susceptibility to parasite infection.Immunol Today1987https://conductscience.org/library/papers/25291681PubMed
122,932,337Dominant suppressive effect of the silent Eb alpha allele on an in vivo T helper cell response under Ed beta Ed alpha region-linked immune response gene control.Eur J Immunol1985https://conductscience.org/library/papers/2932337PubMed
132,978,244The role of suppressor T cells in the expression of immune response gene function.J Mol Cell Immunol1987https://conductscience.org/library/papers/2978244PubMed
143,081,440Isoelectric focusing spectra of anti-bacterial alpha-amylase antibody unique for antigen-induced suppression.Immunology1986https://conductscience.org/library/papers/3081440PubMed
153,086,223How many class II immune response genes? A reappraisal of the evidence.Immunogenetics1986https://conductscience.org/library/papers/3086223PubMed
163,117,681Immune suppression genes control the anti-F antigen response in F1 hybrids and recombinant inbred sets of mice.Immunogenetics1987https://conductscience.org/library/papers/3117681PubMed
173,161,822T helper and T suppressor cells are restricted by the A and E molecules, respectively, in the F antigen system.Immunogenetics1985https://conductscience.org/library/papers/3161822PubMed
184,192,080Immune responses of inbred mice to repeated low doses of antigen: relationship to histocompatibility (H-2) type.Science1970https://conductscience.org/library/papers/4192080PubMed
196,162,794Two distinct high immune response phenotypes are both controlled by H-2 genes mapping K or I-A.Immunogenetics1981https://conductscience.org/library/papers/6162794PubMed
206,184,418Genetic control of the immune response to myoglobin. IX. Overcoming genetic control of antibody response to antigenic sites by increasing the dose of antigen used in immunization.J Immunogenet1982https://conductscience.org/library/papers/6184418PubMed

See all 36 references

Cited by3

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 3 citations.

PMIDTitleJournalYearIn PapersSource
24,715,888Function and Potentials of M. tuberculosis Epitopes.Front Immunol2014https://conductscience.org/library/papers/24715888PubMed
21,714,962H-2 alleles contribute to antigen 85-specific interferon-gamma responses during Mycobacterium tuberculosis infection.Cell Immunol2011https://conductscience.org/library/papers/21714962PubMed
21,257,971A novel human IgA monoclonal antibody protects against tuberculosis.J Immunol2011https://conductscience.org/library/papers/21257971PubMed

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

None in this release

  • Grants
  • Trials
  • Comments and corrections
  • Genes
  • Drugs
  • Data

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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