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Paper · 14517244

Activated EGL-15 FGF receptor promotes protein degradation in muscles of Caenorhabditis elegans.

Szewczyk NJ and 1 other. EMBO J, 2003. Cited by 26 papers, by iCite.

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ConductScience Library. Activated EGL-15 FGF receptor promotes protein degradation in muscles of Caenorhabditis elegans. Papers, record 14517244. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/14517244

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Activated EGL-15 FGF receptor promotes protein degradation in muscles of Caenorhabditis elegans.
PubMed
Journal
EMBO J
PubMed
Year
2003
PubMed
Publication types
Journal Article, Research Support, U.S. Gov't, Non-P.H.S., Research Support, U.S. Gov't, P.H.S.
PubMed
Citations, by iCite
26
iCite
Relative citation ratio, by iCite
0.38
iCite
Revised in PubMed
2018-11-13
PubMed

Authors2

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Szewczyk NJNathaniel J
2Jacobson LALewis A

Source PubMed

Topics9

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Caenorhabditis elegansMajor topic
Caenorhabditis elegans ProteinsMajor topic
Muscle ProteinsMajor topic
Receptors, Fibroblast Growth FactorMajor topic
Animals
Mitogen-Activated Protein Kinases
Muscles
Signal Transduction
ras Proteins

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References60

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
110,806,111Genetic defects in acetylcholine signalling promote protein degradation in muscle cells of Caenorhabditis elegans.J Cell Sci2000https://conductscience.org/library/papers/10806111PubMed
212,024,031Activation of Ras and the mitogen-activated protein kinase pathway promotes protein degradation in muscle cells of Caenorhabditis elegans.Mol Cell Biol2002https://conductscience.org/library/papers/12024031PubMed
310,880,441Caenorhabditis elegans SOS-1 is necessary for multiple RAS-mediated developmental signals.EMBO J2000https://conductscience.org/library/papers/10880441PubMed
410,898,801Gene expression patterns of the fibroblast growth factors and their receptors during myogenesis of rat satellite cells.J Histochem Cytochem2000https://conductscience.org/library/papers/10898801PubMed
511,021,802Regulation of C. elegans life-span by insulinlike signaling in the nervous system.Science2000https://conductscience.org/library/papers/11021802PubMed
611,181,961Cytoplasmic signaling pathways that regulate cardiac hypertrophy.Annu Rev Physiol2001https://conductscience.org/library/papers/11181961PubMed
714,630,038Degradation of transgene-coded and endogenous proteins in the muscles of Caenorhabditis elegans.Biochem Biophys Res Commun2003https://conductscience.org/library/papers/14630038PubMed
83,316,916Protein degradation during endurance exercise and recovery.Med Sci Sports Exerc1987https://conductscience.org/library/papers/3316916PubMed
92,843,547Cell surface fibroblast growth factor and epidermal growth factor receptors are permanently lost during skeletal muscle terminal differentiation in culture.J Cell Biol1988https://conductscience.org/library/papers/2843547PubMed
102,569,235Genetic and pharmacological suppression of oncogenic mutations in ras genes of yeast and humans.Science1989https://conductscience.org/library/papers/2569235PubMed

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Cited by20

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 26 citations.

PMIDTitleJournalYearIn PapersSource
41,849,383The conserved fibroblast growth factor receptor-based signaling is required for dendrite regeneration.Proc Natl Acad Sci U S A2026https://conductscience.org/library/papers/41849383PubMed
33,338,030FGF23 signalling and physiology.J Mol Endocrinol2021https://conductscience.org/library/papers/33338030PubMed
30,782,598A Receptor Tyrosine Kinase Network Regulates Neuromuscular Function in Response to Oxidative Stress in Caenorhabditis elegans.Genetics2019https://conductscience.org/library/papers/30782598PubMed
30,455,409Greater loss of mitochondrial function with ageing is associated with earlier onset of sarcopenia in C. elegans.Aging (Albany NY)2018https://conductscience.org/library/papers/30455409PubMed
28,536,403Anti-Differentiation Effect of Oncogenic Met Receptor in Terminally-Differentiated Myotubes.Biomedicines2015https://conductscience.org/library/papers/28536403PubMed
28,508,547Functional phosphatome requirement for protein homeostasis, networked mitochondria, and sarcomere structure in C. elegans muscle.J Cachexia Sarcopenia Muscle2017https://conductscience.org/library/papers/28508547PubMed
24,285,704Maintenance of muscle myosin levels in adult C. elegans requires both the double bromodomain protein BET-1 and sumoylation.Biol Open2013https://conductscience.org/library/papers/24285704PubMed
24,060,339Knockdown of the C. elegans kinome identifies kinases required for normal protein homeostasis, mitochondrial network structure, and sarcomere structure in muscle.Cell Commun Signal2013https://conductscience.org/library/papers/24060339PubMed
23,908,058Canonical RTK-Ras-ERK signaling and related alternative pathways.WormBook2013https://conductscience.org/library/papers/23908058PubMed
23,900,546The NM23-H1/H2 homolog NDK-1 is required for full activation of Ras signaling in C. elegans.Development2013https://conductscience.org/library/papers/23900546PubMed

1 to 10 of 20

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Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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