Paper · 15030
Lipopolysaccharide-induced suppression of the primary immune response to a thymus-dependent antigen.
Persson U. J Immunol, 1977. Cited by 131 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Lipopolysaccharide-induced suppression of the primary immune response to a thymus-dependent antigen. Papers, record 15030. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/15030
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Lipopolysaccharide-induced suppression of the primary immune response to a thymus-dependent antigen.
- PubMed
Topics17
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Antibody Formation | Major topic |
| B-Lymphocytes | Major topic |
| Immunosuppression Therapy | Major topic |
| Polysaccharides, Bacterial | Major topic |
| Animals | |
| Cell Separation | |
| DNA Replication | |
| Dose-Response Relationship, Immunologic | |
| Erythrocytes | |
| Escherichia coli | |
| Hemolytic Plaque Technique | |
| Lymphocyte Activation | |
| Mice | |
| Spleen | |
| Streptococcus pneumoniae | |
| T-Lymphocytes | |
| Time Factors |
Source PubMed
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Trials0
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Comments and corrections0
PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.
None in this release.
References0
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Cited by27
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 131 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 12,591,899 | Regulatory T cells selectively express toll-like receptors and are activated by lipopolysaccharide. | J Exp Med | 2003 | https://conductscience.org/library/papers/12591899 | PubMed |
| 8,426,125 | In vivo induction of interleukin 10 by anti-CD3 monoclonal antibody or bacterial lipopolysaccharide: differential modulation by cyclosporin A. | J Exp Med | 1993 | https://conductscience.org/library/papers/8426125 | PubMed |
| 7,180,735 | The effect of protease inhibitors on the immune response in mice. | Agents Actions | 1982 | https://conductscience.org/library/papers/7180735 | PubMed |
| 6,783,542 | Increased resistance and depressed delayed-type hypersensitivity to Listeria monocytogenes induced by pretreatment with lipopolysaccharide. | Infect Immun | 1981 | https://conductscience.org/library/papers/6783542 | PubMed |
| 6,715,043 | Mitogenic activities of synthetic lipid A analogs and suppression of mitogenicity of lipid A. | Infect Immun | 1984 | https://conductscience.org/library/papers/6715043 | PubMed |
| 6,459,992 | Lipopolysaccharide-induced activation of suppressor cells: reversal by an agent which alters cyclic nucleotide metabolism. | Immunology | 1982 | https://conductscience.org/library/papers/6459992 | PubMed |
| 6,448,806 | Non-specific inhibitory processes of immunological and mitogenic cellular responses. | Immunology | 1980 | https://conductscience.org/library/papers/6448806 | PubMed |
| 6,413,053 | Menningococcal antigens (MA): a novel immune stimulant in experimental neoplasia. | Cancer Immunol Immunother | 1983 | https://conductscience.org/library/papers/6413053 | PubMed |
| 6,331,888 | In vivo effects of lipopolysaccharide on lymphoid and non-lymphoid cells in the mouse spleen. Reduction of T-lymphocytes and phagocytosis in the inner parts of the periarteriolar lymphocyte sheath. | Cell Tissue Res | 1984 | https://conductscience.org/library/papers/6331888 | PubMed |
| 6,236,151 | T suppressor cells as well as anti-hapten and anti-idiotype B lymphocytes regulate contact sensitivity to oxazolone in mice injected with purified protein derivative from Mycobacterium tuberculosis. | Infect Immun | 1984 | https://conductscience.org/library/papers/6236151 | PubMed |
1 to 10 of 27
See all 27 cited byGenes0
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None in this release.
Drugs0
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None in this release.
Data0
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None in this release.
Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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