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Paper · 16562000

EPITHELIAL CELL PENETRATION AS AN ESSENTIAL STEP IN THE PATHOGENESIS OF BACILLARY DYSENTERY.

Labrec EH and 3 others. J Bacteriol, 1964. Cited by 596 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. EPITHELIAL CELL PENETRATION AS AN ESSENTIAL STEP IN THE PATHOGENESIS OF BACILLARY DYSENTERY. Papers, record 16562000. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/16562000

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
EPITHELIAL CELL PENETRATION AS AN ESSENTIAL STEP IN THE PATHOGENESIS OF BACILLARY DYSENTERY.
PubMed
Journal
J Bacteriol
PubMed
Year
1964
PubMed
Publication types
Journal Article
PubMed
Citations, by iCite
596
iCite
Revised in PubMed
2021-05-26
PubMed

Authors4

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Labrec EHE H
2Schneider HH
3Magnani TJT J
4Formal SBS B

Source PubMed

Topics0

NLM's MeSH indexing of the paper, its major topics first.

None in this release.

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References8

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
214,461,333Experimental Shigella infections. IV. Fluorescent antibody studies of an infection in guinea pigs.J Immunol1961https://conductscience.org/library/papers/14461333PubMed
416,561,983GENETIC HOMOLOGY BETWEEN ESCHERICHIA COLI K-12 AND SALMONELLA.J Bacteriol1962https://conductscience.org/library/papers/16561983PubMed
714,032,030Colonial variants of Shigella flexneri.Acta Microbiol Acad Sci Hung1962https://conductscience.org/library/papers/14032030PubMed
913,783,399Some attributes of virulence in Shigella.Ann N Y Acad Sci1960https://conductscience.org/library/papers/13783399PubMed
1013,816,991The fate of bacteria in the small intestine.J Pathol Bacteriol1960https://conductscience.org/library/papers/13816991PubMed
1113,850,016Mechanisms of the hemodynamic effects of endotoxin.Physiol Rev1960https://conductscience.org/library/papers/13850016PubMed
1213,958,748Experimental Shigella infections. VI. Role of the small intestine in an experimental infection in guinea pigs.J Bacteriol1963https://conductscience.org/library/papers/13958748PubMed
1313,987,307Complement-fixing properties of pepsin-treated rabbit and sheep antibodies.Science1963https://conductscience.org/library/papers/13987307PubMed

Cited by267

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 596 citations.

PMIDTitleJournalYearIn PapersSource
42,818,370Sequential invasion of epithelial and immune cells in Shigellosis.bioRxiv2026https://conductscience.org/library/papers/42818370PubMed
42,779,739Shigella flexneri undergoes niche-specific adaptation during infection.bioRxiv2026https://conductscience.org/library/papers/42779739PubMed
42,459,344Neutrophil kinetics of Shigella infection in Macaca mulatta (Rhesus macaques).Front Cell Infect Microbiol2026https://conductscience.org/library/papers/42459344PubMed
42,212,646Lithocholic acid induces T3SS-dependent formation of invasion-competent Shigella flexneri aggregates.Infect Immun2026https://conductscience.org/library/papers/42212646PubMed
41,755,629The role of the VirB ligand CTP in the molecular mechanism of transcriptional anti-silencing in Shigella flexneri.Nucleic Acids Res2026https://conductscience.org/library/papers/41755629PubMed
41,573,832Differential targeting of human pyroptotic Caspase-5 and Caspase-4 by Shigella OspC2 and OspC3.bioRxiv2025https://conductscience.org/library/papers/41573832PubMed
41,542,648Dual NLRC4 and non-canonical inflammasome signaling drives human GSDMD-mediated killing of Shigella flexneri independently of bacterial cardiolipin.bioRxiv2026https://conductscience.org/library/papers/41542648PubMed
40,787,994A translocation-competent pore is required for Shigella flexneri to escape from the double membrane vacuole during intercellular spread.mBio2025https://conductscience.org/library/papers/40787994PubMed
40,378,153Host PIK3C3 promotes Shigella flexneri spread from cell to cell through vacuole formation.PLoS Pathog2025https://conductscience.org/library/papers/40378153PubMed
40,272,180NLRP11 is required for canonical NLRP3 and non-canonical inflammasome activation during human macrophage infection with mycobacteria.mBio2025https://conductscience.org/library/papers/40272180PubMed

1 to 10 of 267

See all 267 cited by

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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