Paper · 206148
Insulin binding and effects in isolated soleus muscle of lean and obese mice.
Le Marchand-Brustel Y and 2 others. Am J Physiol, 1978. Cited by 248 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Insulin binding and effects in isolated soleus muscle of lean and obese mice. Papers, record 206148. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/206148
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
Topics13
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Glucose | Major topic |
| Insulin | Major topic |
| Muscles | Major topic |
| Obesity | Major topic |
| Animals | |
| Aurothioglucose | |
| Blood Glucose | |
| Deoxyglucose | |
| Dose-Response Relationship, Drug | |
| Male | |
| Mice | |
| Mice, Obese | |
| Receptors, Cell Surface |
Source PubMed
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References0
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Cited by60
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 248 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 41,798,897 | Insulin receptor trafficking and interactions in muscle cells. | J Endocr Soc | 2026 | https://conductscience.org/library/papers/41798897 | PubMed |
| 40,789,980 | The insulin signalling network. | Nat Metab | 2025 | https://conductscience.org/library/papers/40789980 | PubMed |
| 34,921,686 | Human and mouse muscle transcriptomic analyses identify insulin receptor mRNA downregulation in hyperinsulinemia-associated insulin resistance. | FASEB J | 2022 | https://conductscience.org/library/papers/34921686 | PubMed |
| 33,802,826 | Opuntia dillenii (Ker Gawl.) Haw., Seeds Oil Antidiabetic Potential Using In Vivo, In Vitro, In Situ, and Ex Vivo Approaches to Reveal Its Underlying Mechanism of Action. | Molecules | 2021 | https://conductscience.org/library/papers/33802826 | PubMed |
| 33,477,364 | Modulation of Insulin Sensitivity by Insulin-Degrading Enzyme. | Biomedicines | 2021 | https://conductscience.org/library/papers/33477364 | PubMed |
| 32,877,278 | Endoplasmic reticulum stress causes insulin resistance by inhibiting delivery of newly synthesized insulin receptors to the cell surface. | Mol Biol Cell | 2020 | https://conductscience.org/library/papers/32877278 | PubMed |
| 30,542,324 | Role of Endocrine-Disrupting Engineered Nanomaterials in the Pathogenesis of Type 2 Diabetes Mellitus. | Front Endocrinol (Lausanne) | 2018 | https://conductscience.org/library/papers/30542324 | PubMed |
| 30,054,342 | Muscle and adipose tissue insulin resistance: malady without mechanism? | J Lipid Res | 2019 | https://conductscience.org/library/papers/30054342 | PubMed |
| 26,261,819 | Current status and patent prospective of animal models in diabetic research. | Adv Biomed Res | 2015 | https://conductscience.org/library/papers/26261819 | PubMed |
| 11,172,066 | Proteolytic cleavage product of 30-kDa adipocyte complement-related protein increases fatty acid oxidation in muscle and causes weight loss in mice. | Proc Natl Acad Sci U S A | 2001 | https://conductscience.org/library/papers/11172066 | PubMed |
1 to 10 of 60
See all 60 cited byGenes0
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None in this release.
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Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
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- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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