Paper211121
Comparison of calcium-activated, cyclic nucleotide-independent protein kinase and adenosine 3':5'-monophosphate-dependent protein kinase as regards the ability to stimulate glycogen breakdown in vitro.
Kishimoto A and 3 others. J Biochem, 1978. Cited by 19 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Comparison of calcium-activated, cyclic nucleotide-independent protein kinase and adenosine 3':5'-monophosphate-dependent protein kinase as regards the ability to stimulate glycogen breakdown in vitro. Papers, record 211121. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/211121
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
Comparison of calcium-activated, cyclic nucleotide-independent protein kinase and adenosine 3':5'-monophosphate-dependent protein kinase as regards the ability to stimulate glycogen breakdown in vitro.
- PubMed
Topics15
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Calcium | Major topic |
| Cyclic AMP | Major topic |
| Cyclic GMP | Major topic |
| Glycogen | Major topic |
| Protein Kinases | Major topic |
| Adenosine Triphosphate | |
| Animals | |
| Glycogen Synthase | |
| Histones | |
| Liver | |
| Muscles | |
| Phosphorylase Kinase | |
| Protein Kinase Inhibitors | |
| Rabbits | |
| Rats |
Source PubMed
Cited by5
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 19 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 6,948,557 | Alpha 1-Adrenergic stimulation of Ca2+ mobilization without phosphorylase activation in hepatocytes from phosphorylase b kinase-deficient gsd/gsd rats. | Biochem J | 1981 | https://conductscience.org/library/papers/6948557 | PubMed |
| 6,417,664 | Modification of glycogen synthase activity in isolated rat hepatocytes by tumor-promoting phorbol esters: evidence for differential regulation of glycogen synthase and phosphorylase. | Proc Natl Acad Sci U S A | 1983 | https://conductscience.org/library/papers/6417664 | PubMed |
| 3,099,747 | Mechanism of hepatic glycogen synthase inactivation induced by Ca2+-mobilizing hormones. Studies using phospholipase C and phorbol myristate acetate. | Biochem J | 1986 | https://conductscience.org/library/papers/3099747 | PubMed |
| 3,004,415 | Calcium ions and glycogen act synergistically as inhibitors of hepatic glycogen-synthase phosphatase. | Biochem J | 1985 | https://conductscience.org/library/papers/3004415 | PubMed |
| 225,657 | Regulatory and functional compartment of three multifunctional protein kinase systems. | Mol Cell Biochem | 1979 | https://conductscience.org/library/papers/225657 | PubMed |
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- 2026-11-11
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- 2026-09-18
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- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
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- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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