Paper216355
Reversible inhibition of skeletal muscle phosphoprotein phosphatase by ATP, phosphate and fluoride.
Khatra BS and 1 other. Biochem Biophys Res Commun, 1978. Cited by 60 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
Cite this record
ConductScience Library. Reversible inhibition of skeletal muscle phosphoprotein phosphatase by ATP, phosphate and fluoride. Papers, record 216355. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/216355
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Reversible inhibition of skeletal muscle phosphoprotein phosphatase by ATP, phosphate and fluoride.
- PubMed
Topics12
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Adenosine Triphosphate | Major topic |
| Fluorides | Major topic |
| Muscles | Major topic |
| Phosphates | Major topic |
| Phosphoprotein Phosphatases | Major topic |
| Animals | |
| Cations, Divalent | |
| Edetic Acid | |
| Histones | |
| Kinetics | |
| Rabbits | |
| Ribonucleotides |
Source PubMed
Cited by8
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 60 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 36,072,300 | There is no F in APC: Using physiological fluoride-free solutions for high throughput automated patch clamp experiments. | Front Mol Neurosci | 2022 | https://conductscience.org/library/papers/36072300 | PubMed |
| 6,323,956 | Chromatographic characteristics and subcellular localization of synthase phosphatase, phosphorylase phosphatase and histone phosphatase in human polymorphonuclear leukocytes. | Mol Cell Biochem | 1984 | https://conductscience.org/library/papers/6323956 | PubMed |
| 6,278,282 | Inactivation and reactivation of phosphoprotein phosphatase. | Mol Cell Biochem | 1982 | https://conductscience.org/library/papers/6278282 | PubMed |
| 3,025,253 | A simple and rapid preparation of fully phosphorylated and fully dephosphorylated skeletal muscle myosin. Application to the preparation of a phosphorylated LC2-modified artificial isozyme. | J Muscle Res Cell Motil | 1986 | https://conductscience.org/library/papers/3025253 | PubMed |
| 2,848,509 | Fluorine compounds inhibit the conversion of active type-1 protein phosphatases into the ATPMg-dependent form. | Biochem J | 1988 | https://conductscience.org/library/papers/2848509 | PubMed |
| 2,174,099 | Effects of sulfhydryl agents, trifluoperazine, phosphatase inhibitors and tryptic proteolysis on calcineurin isolated from bovine cerebral cortex. | Mol Cell Biochem | 1990 | https://conductscience.org/library/papers/2174099 | PubMed |
| 1,903,935 | Characterization of Ca(2+)-dependent endogenous phosphorylation of 160,000- and 150,000-Dalton proteins of sarcoplasmic reticulum. | Biochem J | 1991 | https://conductscience.org/library/papers/1903935 | PubMed |
| 223,147 | Phosphorylation and inactivation of glycogen synthase by phosphorylase kinase. | Proc Natl Acad Sci U S A | 1979 | https://conductscience.org/library/papers/223147 | PubMed |
Data dates
The date of each source this release read.
- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
None in this release
- Grants
- Trials
- Comments and corrections
- References
- Genes
- Drugs
- Data
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
For software
Read this paper from software.
The record and each of its lists are JSON, twenty rows a page. No key.
GET https://conductscience.org/api/papers/v1/216355
GET https://conductscience.org/api/papers/v1/216355/authors