Paper · 21982925

Distinct mechanisms of DNA repair in mycobacteria and their implications in attenuation of the pathogen growth.

Kurthkoti K and 1 other. Mech Ageing Dev, 2012. Cited by 23 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Distinct mechanisms of DNA repair in mycobacteria and their implications in attenuation of the pathogen growth. Papers, record 21982925. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/21982925

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: Distinct mechanisms of DNA repair in mycobacteria and their implications in attenuation of the pathogen growth.; PubMed
Journal: Mech Ageing Dev; PubMed
Year: 2012; PubMed
Publication types: Journal Article, Review; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/21982925/; PubMed
DOI: https://doi.org/10.1016/j.mad.2011.09.003; PubMed
The journal: https://conductscience.org/library/journal-index/0347227; PubMed
Citations, by iCite: 23; iCite
Relative citation ratio, by iCite: 0.64; iCite
Revised in PubMed: 2012-05-14; PubMed

Authors2

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename	Kind	ORCID iD
1	Kurthkoti K	Krishna
2	Varshney U	Umesh

Source PubMed

Topics17

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
DNA Damage	Major topic
DNA Repair	Major topic
Macrophages	Major topic
Mycobacterium smegmatis	Major topic
Mycobacterium tuberculosis	Major topic
Animals
Anti-Bacterial Agents
Cell Hypoxia
Drug Resistance, Bacterial
Gene Expression Regulation, Bacterial
Genotype
Humans
Microbial Viability
Mutation
Phenotype
Reactive Nitrogen Species
Reactive Oxygen Species

Source PubMed

Grants0

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None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References0

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

None in this release.

Cited by12

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 23 citations.

PMID	Title	Journal	Year	In Papers	Source
40,233,271	Mismatch uracil DNA glycosylase (Mug) is maintained in the Corynebacterium pseudotuberculosis genome and exhibits affinity for uracil but not other types of damage.	Genet Mol Biol	2025	https://conductscience.org/library/papers/40233271	PubMed
39,595,627	Assessing the Impact of Bedaquiline, Clofazimine, and Linezolid on Mycobacterial Genome Integrity.	Biomolecules	2024	https://conductscience.org/library/papers/39595627	PubMed
39,565,350	Genetic stability of Mycobacterium smegmatis under the stress of first-line antitubercular agents.	Elife	2024	https://conductscience.org/library/papers/39565350	PubMed
38,930,573	Sulforaphane Inhibits Oxidative Stress and May Exert Anti-Pyroptotic Effects by Modulating NRF2/NLRP3 Signaling Pathway in Mycobacterium tuberculosis-Infected Macrophages.	Microorganisms	2024	https://conductscience.org/library/papers/38930573	PubMed
26,276,393	Mycobacterium tuberculosis RecG protein but not RuvAB or RecA protein is efficient at remodeling the stalled replication forks: implications for multiple mechanisms of replication restart in mycobacteria.	J Biol Chem	2015	https://conductscience.org/library/papers/26276393	PubMed
25,874,691	Use of Mycobacterium smegmatis deficient in ADP-ribosyltransferase as surrogate for Mycobacterium tuberculosis in drug testing and mutation analysis.	PLoS One	2015	https://conductscience.org/library/papers/25874691	PubMed
25,578,955	The BER necessities: the repair of DNA damage in human-adapted bacterial pathogens.	Nat Rev Microbiol	2015	https://conductscience.org/library/papers/25578955	PubMed
24,800,740	Apurinic/apyrimidinic endonucleases of Mycobacterium tuberculosis protect against DNA damage but are dispensable for the growth of the pathogen in guinea pigs.	PLoS One	2014	https://conductscience.org/library/papers/24800740	PubMed
24,658,131	Either non-homologous ends joining or homologous recombination is required to repair double-strand breaks in the genome of macrophage-internalized Mycobacterium tuberculosis.	PLoS One	2014	https://conductscience.org/library/papers/24658131	PubMed
23,564,173	Biochemical and structural studies of the Mycobacterium tuberculosis O6-methylguanine methyltransferase and mutated variants.	J Bacteriol	2013	https://conductscience.org/library/papers/23564173	PubMed

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Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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