Paper · 2425408

Tolerance induction and maintenance in primed lymphocytes.

Levich JD and 1 other. Surv Immunol Res, 1985. Cited by 1 paper, by iCite.

11 sourcespapers-2026-11-12-0.1.0

Cite this record

ConductScience Library. Tolerance induction and maintenance in primed lymphocytes. Papers, record 2425408. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/2425408

On this page

Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: Tolerance induction and maintenance in primed lymphocytes.; PubMed
Journal: Surv Immunol Res; PubMed
Year: 1985; PubMed
Publication types: Journal Article, Research Support, Non-U.S. Gov't, Research Support, U.S. Gov't, P.H.S.; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/2425408/; PubMed
DOI: https://doi.org/10.1007/bf02918739; PubMed
The journal: https://conductscience.org/library/journal-index/8215669; PubMed
Citations, by iCite: 1; iCite
Relative citation ratio, by iCite: 0.02; iCite
Revised in PubMed: 2019-11-01; PubMed

Authors2

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename	Kind	ORCID iD
1	Levich JD	J D
2	Weigle WO	W O

Source PubMed

Topics12

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Autoimmune Diseases	Major topic
B-Lymphocytes	Major topic
Immune Tolerance	Major topic
T-Lymphocytes	Major topic
Animals
Antibody Formation
Immunologic Memory
Interleukin-2
Ovalbumin
T-Lymphocytes, Helper-Inducer
T-Lymphocytes, Regulatory
gamma-Globulins

Source PubMed

Grants2

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes it	Agency	Country	NIH project	Stated by
AI07007	NIAID NIH HHS	United States		PubMed
RRO-5514	NCRR NIH HHS	United States		PubMed

Source PubMed

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References33

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

Order	PMID	Title	Journal	Year	In Papers	Source
1	6,166,703	In vitro tolerance induction of primed, IgD-negative murine spleen cells.	J Exp Med	1981	https://conductscience.org/library/papers/6166703	PubMed
2	6,197,463	Establishment and functional analysis of a cloned, antigen-specific suppressor effector T cell line.	J Immunol	1984	https://conductscience.org/library/papers/6197463	PubMed
3	6,153,675	Maintenance of immunologic unresponsiveness to human gamma-globulin: evidence for irreversible inactivation in B lymphocytes.	J Immunol	1980	https://conductscience.org/library/papers/6153675	PubMed
4	47,879	Preventive effect of hapten-reactive thymus-derived helper lymphocytes on the tolerance induction in hapten-specific precursors of antibody-forming cells.	J Immunol	1975	https://conductscience.org/library/papers/47879	PubMed
5	6,168,689	Establishment of unresponsiveness in primed B lymphocytes in vivo.	J Immunol	1981	https://conductscience.org/library/papers/6168689	PubMed
6	6,310,611	Magnitude of response of histocompatibility-restricted T-cell clones is a function of the product of the concentrations of antigen and Ia molecules.	Proc Natl Acad Sci U S A	1983	https://conductscience.org/library/papers/6310611	PubMed
7	6,194,243	T cell regulation of b cell activation. Cloned Lyt-1+2-T suppressor cells inhibit the major histocompatibility complex-restricted interaction of T helper cells with B cells and/or accessory cells.	J Exp Med	1983	https://conductscience.org/library/papers/6194243	PubMed
8	58,041	Specific suppression of the immune response by HGG tolerant spleen cells. I. Parameters affecting the level of suppression.	J Immunol	1976	https://conductscience.org/library/papers/58041	PubMed
9	6,234,354	Antigen-reactive cloned helper T cells. II. Exposure of murine cloned helper T cells to IL 2-containing supernatant induces unresponsiveness to antigenic restimulation and inhibits lymphokine production after antigenic stimulation.	J Immunol	1984	https://conductscience.org/library/papers/6234354	PubMed
10	25,291,390	Antigen specific suppression of T-cell responses - the veto concept.	Immunol Today	1985	https://conductscience.org/library/papers/25291390	PubMed

1 to 10 of 33

See all 33 references

Cited by0

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 1 citation.

None in this release.

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

For software

Read this paper from software.

The record and each of its lists are JSON, twenty rows a page. No key.

GET https://conductscience.org/api/papers/v1/2425408

GET https://conductscience.org/api/papers/v1/2425408/references?page=2

API reference For AI assistants