Paper26770
Antidotal effects of dimethyl sulphoxide against paracetamol-, bromobenzene-, and thioacetamide-induced hepatotoxicity.
Siegers CP. J Pharm Pharmacol, 1978. Cited by 38 papers, by iCite.
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ConductScience Library. Antidotal effects of dimethyl sulphoxide against paracetamol-, bromobenzene-, and thioacetamide-induced hepatotoxicity. Papers, record 26770. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/26770
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Antidotal effects of dimethyl sulphoxide against paracetamol-, bromobenzene-, and thioacetamide-induced hepatotoxicity.
- PubMed
Topics10
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Acetamides | Major topic |
| Acetaminophen | Major topic |
| Bromobenzenes | Major topic |
| Dimethyl Sulfoxide | Major topic |
| Thioacetamide | Major topic |
| Animals | |
| Antidotes | |
| Liver | |
| Male | |
| Mice |
Source PubMed
Cited by9
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 38 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 35,221,288 | Roles of Cofactors in Drug-Induced Liver Injury: Drug Metabolism and Beyond. | Drug Metab Dispos | 2022 | https://conductscience.org/library/papers/35221288 | PubMed |
| 31,191,316 | Dimethyl Sulfoxide Decreases Levels of Oxylipin Diols in Mouse Liver. | Front Pharmacol | 2019 | https://conductscience.org/library/papers/31191316 | PubMed |
| 31,007,174 | Animal models of drug-induced liver injury. | Biochim Biophys Acta Mol Basis Dis | 2019 | https://conductscience.org/library/papers/31007174 | PubMed |
| 30,654,094 | The inhibitor of glycerol 3-phosphate acyltransferase FSG67 blunts liver regeneration after acetaminophen overdose by altering GSK3β and Wnt/β-catenin signaling. | Food Chem Toxicol | 2019 | https://conductscience.org/library/papers/30654094 | PubMed |
| 19,236,887 | Role of oxidative stress in cadmium toxicity and carcinogenesis. | Toxicol Appl Pharmacol | 2009 | https://conductscience.org/library/papers/19236887 | PubMed |
| 11,565,617 | Bupivacaine hydrochloride induces muscle fiber necrosis and hydroxyl radical formation-dimethyl sulphoxide reduces hydroxyl radical formation. | Neurochem Res | 2001 | https://conductscience.org/library/papers/11565617 | PubMed |
| 6,343,056 | Paracetamol overdosage. Pharmacological considerations and clinical management. | Drugs | 1983 | https://conductscience.org/library/papers/6343056 | PubMed |
| 6,249,238 | Inhibition of the hepatotoxicity of paracetamol and its irreversible binding to rat liver microsomal protein. | Arch Toxicol | 1980 | https://conductscience.org/library/papers/6249238 | PubMed |
| 229,786 | Effect of dithiocarb and dimethyl sulfoxide on irreversible binding of 14C-bromobenzene to rat liver microsomal protein. | Arch Toxicol | 1979 | https://conductscience.org/library/papers/229786 | PubMed |
Data dates
The date of each source this release read.
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- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
None in this release
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- Drugs
- Data
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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