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Paper · 29367285

Che-1 is targeted by c-Myc to sustain proliferation in pre-B-cell acute lymphoblastic leukemia.

Folgiero V and 17 others. EMBO Rep, 2018. Cited by 24 papers, by iCite.

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ConductScience Library. Che-1 is targeted by c-Myc to sustain proliferation in pre-B-cell acute lymphoblastic leukemia. Papers, record 29367285. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/29367285

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Che-1 is targeted by c-Myc to sustain proliferation in pre-B-cell acute lymphoblastic leukemia.
PubMed
Journal
EMBO Rep
PubMed
Year
2018
PubMed
Publication types
Journal Article, Research Support, Non-U.S. Gov't
PubMed
Citations, by iCite
24
iCite
Relative citation ratio, by iCite
0.72
iCite
Revised in PubMed
2022-03-21
PubMed

Authors18

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Folgiero VValentina
2Sorino CCristinahttps://orcid.org/0000-0002-6100-289X
3Pallocca MMatteo
4De Nicola FFrancesca
5Goeman FFrauke
6Bertaina VValentina
7Strocchio LLuisa
8Romania PPaolo
9Pitisci AAngela
10Iezzi SSimona

1 to 10 of 18Source PubMed

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Topics13

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Apoptosis Regulatory ProteinsMajor topic
Precursor B-Cell Lymphoblastic Leukemia-LymphomaMajor topic
Precursor Cell Lymphoblastic Leukemia-LymphomaMajor topic
Proto-Oncogene Proteins c-mycMajor topic
Repressor ProteinsMajor topic
Cell Line, Tumor
Cell Proliferation
DNA-Binding Proteins
Gene Expression Regulation, Leukemic
High-Throughput Nucleotide Sequencing
Humans
Neoplasm Recurrence, Local
Promoter Regions, Genetic

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References50

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
111,071,758The murine gene, Traube, is essential for the growth of preimplantation embryos.Dev Biol2000https://conductscience.org/library/papers/11071758PubMed
214,627,703AATF inhibits aberrant production of amyloid beta peptide 1-42 by interacting directly with Par-4.J Biol Chem2004https://conductscience.org/library/papers/14627703PubMed
322,120,020A subset of chronic lymphocytic leukemia patients display reduced levels of PARP1 expression coupled with a defective irradiation-induced apoptosis.Exp Hematol2012https://conductscience.org/library/papers/22120020PubMed
412,450,794Che-1 affects cell growth by interfering with the recruitment of HDAC1 by Rb.Cancer Cell2002https://conductscience.org/library/papers/12450794PubMed
512,808,131A global transcriptional regulatory role for c-Myc in Burkitt's lymphoma cells.Proc Natl Acad Sci U S A2003https://conductscience.org/library/papers/12808131PubMed
625,060,305C-myc protein expression in B-cell acute lymphoblastic leukemia, prognostic significance?Leuk Res2014https://conductscience.org/library/papers/25060305PubMed
723,021,215Transcriptional amplification in tumor cells with elevated c-Myc.Cell2012https://conductscience.org/library/papers/23021215PubMed
825,770,584Che-1-induced inhibition of mTOR pathway enables stress-induced autophagy.EMBO J2015https://conductscience.org/library/papers/25770584PubMed
924,799,436deepTools: a flexible platform for exploring deep-sequencing data.Nucleic Acids Res2014https://conductscience.org/library/papers/24799436PubMed
1021,602,269CENTDIST: discovery of co-associated factors by motif distribution.Nucleic Acids Res2011https://conductscience.org/library/papers/21602269PubMed

1 to 10 of 50

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Cited by20

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 24 citations.

PMIDTitleJournalYearIn PapersSource
42,441,112Targeting AATF reprograms the tumor microenvironment and suppresses hepatocellular carcinoma via MIR100HG-TGF-β signaling.Mol Ther Oncol2026https://conductscience.org/library/papers/42441112PubMed
41,872,157AATF supports proliferation of glioblastoma cells by sustaining mitochondrial respiration through an NRF-1-dependent mechanism.Cell Death Dis2026https://conductscience.org/library/papers/41872157PubMed
38,926,853Enhancer engagement sustains oncogenic transformation and progression of B-cell precursor acute lymphoblastic leukemia.J Exp Clin Cancer Res2024https://conductscience.org/library/papers/38926853PubMed
37,767,562Profiling the activity of the para-caspase MALT1 in B-cell acute lymphoblastic leukemia for potential targeted therapeutic application.Haematologica2024https://conductscience.org/library/papers/37767562PubMed
37,435,061AATF/Che-1 RNA polymerase II binding protein overexpression reduces the anti-tumor NK-cell cytotoxicity through activating receptors modulation.Front Immunol2023https://conductscience.org/library/papers/37435061PubMed
35,929,179AATF/Che-1 localizes to paraspeckles and suppresses R-loops accumulation and interferon activation in Multiple Myeloma.EMBO J2022https://conductscience.org/library/papers/35929179PubMed
34,785,906AATF is Overexpressed in Human Head and Neck Squamous Cell Carcinoma and Regulates STAT3/Survivin Signaling.Onco Targets Ther2021https://conductscience.org/library/papers/34785906PubMed
34,266,450CK2-mediated phosphorylation of Che-1/AATF is required for its pro-proliferative activity.J Exp Clin Cancer Res2021https://conductscience.org/library/papers/34266450PubMed
33,737,337GD2 redirected CAR T and activated NK-cell-mediated secretion of IFNγ overcomes MYCN-dependent IDO1 inhibition, contributing to neuroblastoma cell immune escape.J Immunother Cancer2021https://conductscience.org/library/papers/33737337PubMed
33,186,461Che-1/AATF-induced transcriptionally active chromatin promotes cell proliferation in multiple myeloma.Blood Adv2020https://conductscience.org/library/papers/33186461PubMed

1 to 10 of 20

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Genes2

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

GeneNCBI Gene IDLinked byIn GenesSource
AATF26,574PubTator's text mininghttps://conductscience.org/library/genes/26574NCBI Gene
MYC4,609PubTator's text mininghttps://conductscience.org/library/genes/4609NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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