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Paper301441

Discrimination of suppressor T cells of humoral and cell-mediated immunity by anti-Ly and anti-Ia sera.

Ramshaw IA and 3 others. Cell Immunol, 1977. Cited by 92 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Discrimination of suppressor T cells of humoral and cell-mediated immunity by anti-Ly and anti-Ia sera. Papers, record 301441. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/301441

Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Discrimination of suppressor T cells of humoral and cell-mediated immunity by anti-Ly and anti-Ia sera.
PubMed
Journal
Cell Immunol
PubMed
Year
1977
PubMed
Publication types
Journal Article, Research Support, U.S. Gov't, P.H.S.
PubMed
Citations, by iCite
92
iCite
Relative citation ratio, by iCite
2.14
iCite
Revised in PubMed
2021-12-03
PubMed

Authors4

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Ramshaw IAI A
2McKenzie IFI F
3Bretscher PAP A
4Parish CRC R

Source PubMed

Topics12

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Antibody FormationMajor topic
Histocompatibility AntigensMajor topic
Hypersensitivity, DelayedMajor topic
T-LymphocytesMajor topic
Animals
Female
Immunosuppression Therapy
Mice
Mice, Inbred CBA
Phenotype
Spleen
Surface Properties

Source PubMed

Cited by19

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 92 citations.

PMIDTitleJournalYearIn PapersSource
36,405,696Analyzing some concepts of immune regulation of the last three decades: Fostering greater research resilience despite the information overload. A personal view.Front Immunol2022https://conductscience.org/library/papers/36405696PubMed
31,231,378On Analyzing How the Th1/Th2 Phenotype of an Immune Response Is Determined: Classical Observations Must Not Be Ignored.Front Immunol2019https://conductscience.org/library/papers/31231378PubMed
24,684,592On the mechanism determining the TH1/TH2 phenotype of an immune response, and its pertinence to strategies for the prevention, and treatment, of certain infectious diseases.Scand J Immunol2014https://conductscience.org/library/papers/24684592PubMed
18,188,562Different immune correlates associated with tumor progression and regression: implications for prevention and treatment of cancer.Cancer Immunol Immunother2008https://conductscience.org/library/papers/18188562PubMed
9,826,349Mycobacterial dose defines the Th1/Th2 nature of the immune response independently of whether immunization is administered by the intravenous, subcutaneous, or intradermal route.Infect Immun1998https://conductscience.org/library/papers/9826349PubMed
6,448,222The surface phenotype of a suppressor cell of delayed-type hypersensitivity in the mouse.Immunology1980https://conductscience.org/library/papers/6448222PubMed
6,447,751Immunological regulation of experimental cutaneous leishmaniasis. III. Nature and significance of specific suppression of cell-mediated immunity in mice highly susceptible to Leishmania tropica.J Exp Med1980https://conductscience.org/library/papers/6447751PubMed
6,231,248Immunoregulation of genetically controlled acquired responses to Leishmania donovani infection in mice: demonstration and characterization of suppressor T cells in noncure mice.Infect Immun1984https://conductscience.org/library/papers/6231248PubMed
6,228,519Induction and Ly phenotype of suppressor T cells in mice during primary infection with dengue virus.Immunology1984https://conductscience.org/library/papers/6228519PubMed
6,214,501Concanavalin-induced suppressor cells: characterization on the basis of corticosteroid and radiation sensitivity.Immunology1982https://conductscience.org/library/papers/6214501PubMed
6,199,292Allospecific suppression without requirement for participation of Ly 2+ cells.Immunology1984https://conductscience.org/library/papers/6199292PubMed
6,157,084Antigen specific T cell factors.Mol Cell Biochem1980https://conductscience.org/library/papers/6157084PubMed
6,154,762Delayed-type hypersensitivity to influenza virus. Induction of antigen-specific suppressor T cells for delayed-type hypersensitivity to hemagglutinin during influenza virus infection in mice.J Exp Med1980https://conductscience.org/library/papers/6154762PubMed
3,921,652Therapy of disseminated murine leukemia with cyclophosphamide and immune Lyt-1+,2- T cells. Tumor eradication does not require participation of cytotoxic T cells.J Exp Med1985https://conductscience.org/library/papers/3921652PubMed
3,349,644
Augmentation of delayed-type hypersensitivity to high dose sheep erythrocytes by cyclosporin A in the mouse: influence of drug dosage and route of administration and analysis of spleen cell populations.
Clin Exp Immunol1988https://conductscience.org/library/papers/3349644PubMed
2,932,385Suppression of induced erythrocyte autoantibodies is dependent on Lyt 1 cells.Immunology1985https://conductscience.org/library/papers/2932385PubMed
2,142,482Characterization of Candida albicans mannan-induced, mannan-specific delayed hypersensitivity suppressor cells.Infect Immun1990https://conductscience.org/library/papers/2142482PubMed
309,853Distinction between suppressors of the delayed-type hypersensitivity and the humoral response to sheep erythrocytes.Immunology1978https://conductscience.org/library/papers/309853PubMed
156,197Inhibition of humoral and cell-mediated immune responses in man by distinct suppressor cell systems.J Clin Invest1979https://conductscience.org/library/papers/156197PubMed

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

None in this release

  • Grants
  • Trials
  • Comments and corrections
  • References
  • Genes
  • Drugs
  • Data

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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