Paper · 3134499

T cell receptors of autoimmune mice: functional and molecular analysis of novel T cell subsets in C3H-gld/gld mice.

Yui K and 2 others. Immunol Res, 1988. Cited by 10 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. T cell receptors of autoimmune mice: functional and molecular analysis of novel T cell subsets in C3H-gld/gld mice. Papers, record 3134499. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/3134499

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: T cell receptors of autoimmune mice: functional and molecular analysis of novel T cell subsets in C3H-gld/gld mice.; PubMed
Journal: Immunol Res; PubMed
Year: 1988; PubMed
Publication types: Journal Article, Research Support, U.S. Gov't, P.H.S., Review; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/3134499/; PubMed
DOI: https://doi.org/10.1007/bf02918099; PubMed
The journal: https://conductscience.org/library/journal-index/8611087; PubMed
Citations, by iCite: 10; iCite
Relative citation ratio, by iCite: 0.14; iCite
Revised in PubMed: 2019-10-29; PubMed

Authors3

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename
1	Yui K	K
2	Hashimoto Y	Y
3	Greene MI	M I

Source PubMed

Topics10

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Autoimmune Diseases	Major topic
Receptors, Antigen, T-Cell	Major topic
Animals
Lymphocyte Activation
Lymphokines
Mice
Mice, Mutant Strains
Receptors, Immunologic
Receptors, Interleukin-2
T-Lymphocytes

Source PubMed

Grants4

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes it	Agency	Country	NIH project	Stated by	Source
AI23244	NIAID NIH HHS	United States	R01AI023244	PubMed and NIH	NIH RePORTER
AI24019	NIAID NIH HHS	United States	R01AI024019	PubMed and NIH	NIH RePORTER
AR38187	NIAMS NIH HHS	United States	R23AR038187	PubMed and NIH	NIH RePORTER
			R23AI023244	NIH	NIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References61

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

Order	PMID	Title	Journal	Year	In Papers	Source
1	6,601,004	The role of accessory cells in polyclonal T cell activation. I. Both induction of interleukin 2 production and of interleukin 2 responsiveness by concanavalin A are accessory cell dependent.	Eur J Immunol	1983	https://conductscience.org/library/papers/6601004	PubMed
2	3,928,747	Functional analysis of T cell subsets from mice bearing the lpr gene.	J Immunol	1985	https://conductscience.org/library/papers/3928747	PubMed
3	6,144,707	The proliferating cells in autoimmune MRL/lpr mice lack L3T4, an antigen on "helper" T cells that is involved in the response to class II major histocompatibility antigens.	J Immunol	1984	https://conductscience.org/library/papers/6144707	PubMed
4	2,441,398	T-cell receptor genes in autoimmune mice: T-cell subsets have unexpected T-cell receptor gene programs.	Proc Natl Acad Sci U S A	1987	https://conductscience.org/library/papers/2441398	PubMed
5	6,184,304	Cloned T lymphocytes and monoclonal antibodies as probes for cell surface molecules active in T cell-mediated cytolysis.	Immunol Rev	1982	https://conductscience.org/library/papers/6184304	PubMed
6	87,465	Properties and applications of monoclonal antibodies directed against determinants of the Thy-1 locus.	J Immunol	1979	https://conductscience.org/library/papers/87465	PubMed
7	6,442,264	Cell surface antigen expression on thymocytes: development and phenotypic differentiation of intrathymic subsets.	Immunol Rev	1984	https://conductscience.org/library/papers/6442264	PubMed
8	315,430	Ligand-activated T cell growth factor-induced proliferation: absorption of T cell growth factor by activated T cells.	J Immunol	1979	https://conductscience.org/library/papers/315430	PubMed
9	6,984,061	Possible involvement of the OKT4 molecule in T cell recognition of class II HLA antigens. Evidence from studies of cytotoxic T lymphocytes specific for SB antigens.	J Exp Med	1982	https://conductscience.org/library/papers/6984061	PubMed
10	6,454,755	Antibodies to membrane structures that distinguish suppressor/cytotoxic and helper T lymphocyte subpopulations block the mixed leukocyte reaction in man.	J Exp Med	1981	https://conductscience.org/library/papers/6454755	PubMed

1 to 10 of 61

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Cited by3

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 10 citations.

PMID	Title	Journal	Year	In Papers	Source
9,479,571	Fas- and perforin-independent mechanism of cytotoxic T lymphocyte.	Immunol Res	1998	https://conductscience.org/library/papers/9479571	PubMed
2,563,589	Major histocompatibility complex class I-specific cytolytic T cells, derived from gld mice, lacking Thy-1, CD4, and CD8.	Proc Natl Acad Sci U S A	1989	https://conductscience.org/library/papers/2563589	PubMed
1,534,828	CD4-CD8+ T-cell receptor-alpha beta+ major histocompatibility complex class-II-specific T-cell clones isolated from aged athymic mice.	Immunol Res	1992	https://conductscience.org/library/papers/1534828	PubMed

Genes0

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None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

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ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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