Paper · 322367

Idiotypes as variable region markers: analogies between receptors on phosphorylcholine-specific T and B lymphocytes.

Cosenza H and 2 others. Immunol Rev, 1977. Cited by 166 papers, by iCite.

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ConductScience Library. Idiotypes as variable region markers: analogies between receptors on phosphorylcholine-specific T and B lymphocytes. Papers, record 322367. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/322367

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: Idiotypes as variable region markers: analogies between receptors on phosphorylcholine-specific T and B lymphocytes.; PubMed
Journal: Immunol Rev; PubMed
Year: 1977; PubMed
Publication types: Journal Article, Review; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/322367/; PubMed
DOI: https://doi.org/10.1111/j.1600-065x.1977.tb00366.x; PubMed
The journal: https://conductscience.org/library/journal-index/7702118; PubMed
Citations, by iCite: 166; iCite
Relative citation ratio, by iCite: 3.87; iCite
Revised in PubMed: 2021-12-03; PubMed

Authors3

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename
1	Cosenza H	H
2	Julius MH	M H
3	Augustin AA	A A

Source PubMed

Topics18

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
B-Lymphocytes	Major topic
Binding Sites, Antibody	Major topic
Choline	Major topic
Immunoglobulin Variable Region	Major topic
Phosphorylcholine	Major topic
T-Lymphocytes	Major topic
Animals
Animals, Newborn
Autoantibodies
Female
Haptens
Immunity, Cellular
Immunosuppression Therapy
Maternal-Fetal Exchange
Mice
Mice, Inbred BALB C
Pregnancy
Spleen

Source PubMed

Grants0

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Trials0

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Comments and corrections0

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References0

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Cited by50

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 166 citations.

PMID	Title	Journal	Year	In Papers	Source
22,457,936	Genetics of the anti-dextran B512 and the autoanti-idiotypic response: codominant expression in F1 hybrids and dichotomy of response and allotype-linked idiotype.	Immunogenetics	1980	https://conductscience.org/library/papers/22457936	PubMed
7,049,456	The assessment of anti-idiopathic antibodies as effective immunoregulatory probes in vivo.	Clin Exp Immunol	1982	https://conductscience.org/library/papers/7049456	PubMed
7,031,652	Suppression of a "recurrent" idiotype results in profound alterations of the whole B-cell compartment.	Proc Natl Acad Sci U S A	1981	https://conductscience.org/library/papers/7031652	PubMed
7,028,912	Expression of an idiotype (Id-460) during in vivo anti-dinitrophenyl antibody responses. II. Transient idiotypic dominance.	J Exp Med	1981	https://conductscience.org/library/papers/7028912	PubMed
7,014,422	Inheritance patterns of idiotype expression: maternal-fetal immune regulatory networks.	Immunogenetics	1981	https://conductscience.org/library/papers/7014422	PubMed
6,985,352	Recurrent idiotopes and internal images.	EMBO J	1982	https://conductscience.org/library/papers/6985352	PubMed
6,972,994	Generation of idiotype-specific T cell help through network perturbation.	J Exp Med	1981	https://conductscience.org/library/papers/6972994	PubMed
6,968,810	Late clonal selection and expansion of the TEPC-15 germ-line specificity.	J Exp Med	1980	https://conductscience.org/library/papers/6968810	PubMed
6,966,320	Selective suppression of the major idiotypic component of an antihapten response by soluble T cell-derived factors with idiotypic or anti-idiotypic receptors.	J Exp Med	1980	https://conductscience.org/library/papers/6966320	PubMed
6,854,211	Regulation of natural antiallotype antibody responses by idiotype network-induced auto-antiidiotypic antibodies.	J Exp Med	1983	https://conductscience.org/library/papers/6854211	PubMed

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Drugs0

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None in this release.

Data0

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Data dates

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PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

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ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

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ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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