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Paper324902

Inhibition of antibody responses by cells from mice treated with picryl sulphonic acid.

Thomas WR and 1 other. Immunology, 1977. Cited by 11 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Inhibition of antibody responses by cells from mice treated with picryl sulphonic acid. Papers, record 324902. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/324902

Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Inhibition of antibody responses by cells from mice treated with picryl sulphonic acid.
PubMed
Journal
Immunology
PubMed
Year
1977
PubMed
Publication types
Journal Article
PubMed
Citations, by iCite
11
iCite
Relative citation ratio, by iCite
0.3
iCite
Revised in PubMed
2021-12-03
PubMed

Authors2

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Thomas WRW R
2Asherson GLG L

Source PubMed

Topics15

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Antibody FormationMajor topic
NitrobenzenesMajor topic
Trinitrobenzenesulfonic AcidMajor topic
Animals
Antigens
Dose-Response Relationship, Immunologic
Hemolytic Plaque Technique
Immunoglobulin G
Immunosuppression Therapy
Lymphocyte Transfusion
Male
Mice
Nitrophenols
Time Factors
Transplantation, Homologous

Source PubMed

References13

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
14,123,957The immune response against hapten-autologous protein conjugates in the mouse. I. Specificity of antibodies produced during the primary response against dinitrophenylated mouse serum albumin.Eur J Immunol1973https://conductscience.org/library/papers/4123957PubMed
24,120,095Immunological tolerance to a hapten. I. Induction and maintenance of tolerance to trinitrophenyl with trinitrobenzene sulfonic acid.J Exp Med1973https://conductscience.org/library/papers/4120095PubMed
34,583,095Depression of the T cell phenomenon of contact sensitivity by T cells from unresponsive mice.Nature1973https://conductscience.org/library/papers/4583095PubMed
44,545,896Cell-to-cell interaction in the immune response. X. T-cell-dependent suppression in tolerant mice.J Exp Med1974https://conductscience.org/library/papers/4545896PubMed
54,542,648Contact sensitivity in the mouse. XI. Movement of T blasts in the draining lymph nodes to sites of inflammation.Immunology1973https://conductscience.org/library/papers/4542648PubMed
64,997,686An immunological and chromatographic comparison of human intrinsic factor and the acid-stable gastric esterase VI A.Clin Exp Immunol1971https://conductscience.org/library/papers/4997686PubMed
74,941,661Lymphocyte activation. I. Response of T and B lymphocytes to phytomitogens.Clin Exp Immunol1971https://conductscience.org/library/papers/4941661PubMed
84,921,783Specific inhibition of an anti-hapten immune response by chemical modifications of cells.J Immunol1970https://conductscience.org/library/papers/4921783PubMed
91,101,472The role of specific suppressor T cells in immune tolerance.Transplant Rev1975https://conductscience.org/library/papers/1101472PubMed
101,079,199Permanent hapten-specific tolerance in B lymphocytes.Immunology1975https://conductscience.org/library/papers/1079199PubMed
115,355,110Antitrinitrophenyl (TNP) plaque assay. Primary response of Balb/c mice to soluble and particulate immunogen.Proc Soc Exp Biol Med1969https://conductscience.org/library/papers/5355110PubMed
125,653,817Further improvements in the plaque technique for detecting single antibody-forming cells.Immunology1968https://conductscience.org/library/papers/5653817PubMed
135,925,708Immunogenicity of trinitrophenyl-hemocyanin: production of primary and secondary anti-hapten precipitins.J Immunol1966https://conductscience.org/library/papers/5925708PubMed

Cited by5

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 11 citations.

PMIDTitleJournalYearIn PapersSource
7,380,471Hapten-specific unresponsiveness in mice. II. Fate and distribution of 14C-TNBSA injected at tolerogenic doses.Immunology1980https://conductscience.org/library/papers/7380471PubMed
721,137Induction of contact sensitivity. Selective induction of delayed hypersensitivity by the injection of cells from draining lymph nodes into the footpads of normal recipients.Immunology1978https://conductscience.org/library/papers/721137PubMed
312,271Suppression of antibody responses by cells from mice painted with picryl chloride.Immunology1979https://conductscience.org/library/papers/312271PubMed
86,508Hapten specific unresponsiveness in mice. I. Characteristics of unresponsiveness induced by 2,4,6-trinitrobenzene sulphonic acid.Immunology1979https://conductscience.org/library/papers/86508PubMed
86,500Split unresponsiveness to trinitrophenyl (TNP) determinant. Suppression of anti-TNP antibody responses by sensitization with picryl chloride.Immunology1978https://conductscience.org/library/papers/86500PubMed

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

None in this release

  • Grants
  • Trials
  • Comments and corrections
  • Genes
  • Drugs
  • Data

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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