Paper · 340309
Alteration of islet cell populations in spontaneously diabetic mice.
Baetens D and 5 others. Diabetes, 1978. Cited by 148 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Alteration of islet cell populations in spontaneously diabetic mice. Papers, record 340309. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/340309
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
Topics13
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Diabetes Mellitus | Major topic |
| Islets of Langerhans | Major topic |
| Animals | |
| Disease Models, Animal | |
| Fluorescent Antibody Technique | |
| Glucagon | |
| Insulin | |
| Mice | |
| Mice, Inbred C57BL | |
| Mice, Obese | |
| Obesity | |
| Peptides | |
| Somatostatin |
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References0
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Cited by61
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 148 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 41,356,183 | Long-acting management of diabetes and associated complications using an injectable thermosensitive hydrogel incorporating IgG-conjugated GLP-1RA. | Theranostics | 2026 | https://conductscience.org/library/papers/41356183 | PubMed |
| 39,797,759 | E3 ligase substrate adaptor SPOP fine-tunes the UPR of pancreatic β cells. | Genes Dev | 2025 | https://conductscience.org/library/papers/39797759 | PubMed |
| 38,298,268 | Pharmacologic inhibition of somatostatin receptor 2 to restore glucagon counterregulation in diabetes. | Front Pharmacol | 2023 | https://conductscience.org/library/papers/38298268 | PubMed |
| 37,401,013 | Contribution of animal models to diabetes research: Its history, significance, and translation to humans. | J Diabetes Investig | 2023 | https://conductscience.org/library/papers/37401013 | PubMed |
| 37,020,955 | Steroidogenic factor 1 protects mice from obesity-induced glucose intolerance via improving glucose-stimulated insulin secretion by beta cells. | iScience | 2023 | https://conductscience.org/library/papers/37020955 | PubMed |
| 35,167,496 | Notch-mediated Ephrin signaling disrupts islet architecture and β cell function. | JCI Insight | 2022 | https://conductscience.org/library/papers/35167496 | PubMed |
| 34,231,467 | Reduced synchroneity of intra-islet Ca2+ oscillations in vivo in Robo-deficient β cells. | Elife | 2021 | https://conductscience.org/library/papers/34231467 | PubMed |
| 33,993,642 | β-Cell failure in diabetes: Common susceptibility and mechanisms shared between type 1 and type 2 diabetes. | J Diabetes Investig | 2021 | https://conductscience.org/library/papers/33993642 | PubMed |
| 33,586,491 | Pancreatic β-cell heterogeneity in health and diabetes: classes, sources, and subtypes. | Am J Physiol Endocrinol Metab | 2021 | https://conductscience.org/library/papers/33586491 | PubMed |
| 32,739,864 | A surrogate of Roux-en-Y gastric bypass (the enterogastro anastomosis surgery) regulates multiple beta-cell pathways during resolution of diabetes in ob/ob mice. | EBioMedicine | 2020 | https://conductscience.org/library/papers/32739864 | PubMed |
1 to 10 of 61
See all 61 cited byGenes0
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None in this release.
Data0
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Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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