Paper340960
D-glucose inhibits potassium efflux from pancreatic islet cells.
Henquin JC. Nature, 1978. Cited by 221 papers, by iCite.
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ConductScience Library. D-glucose inhibits potassium efflux from pancreatic islet cells. Papers, record 340960. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/340960
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
Topics9
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Glucose | Major topic |
| Islets of Langerhans | Major topic |
| Potassium | Major topic |
| Calcium | |
| Cell Membrane Permeability | |
| Insulin | |
| Mannoheptulose | |
| Membrane Potentials | |
| Molecular Conformation |
Source PubMed
Cited by63
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 221 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 37,815,796 | KATP Channels and the Metabolic Regulation of Insulin Secretion in Health and Disease: The 2022 Banting Medal for Scientific Achievement Award Lecture. | Diabetes | 2023 | https://conductscience.org/library/papers/37815796 | PubMed |
| 34,199,454 | What Is the Metabolic Amplification of Insulin Secretion and Is It (Still) Relevant? | Metabolites | 2021 | https://conductscience.org/library/papers/34199454 | PubMed |
| 27,938,490 | Transgenic Expression of Glucagon-Like Peptide-1 (GLP-1) and Activated Muscarinic Receptor (M3R) Significantly Improves Pig Islet Secretory Function. | Cell Transplant | 2017 | https://conductscience.org/library/papers/27938490 | PubMed |
| 26,239,056 | Type 2 Diabetes-Associated K+ Channel TALK-1 Modulates β-Cell Electrical Excitability, Second-Phase Insulin Secretion, and Glucose Homeostasis. | Diabetes | 2015 | https://conductscience.org/library/papers/26239056 | PubMed |
| 16,321,791 | Glucose-sensing mechanisms in pancreatic beta-cells. | Philos Trans R Soc Lond B Biol Sci | 2005 | https://conductscience.org/library/papers/16321791 | PubMed |
| 12,830,383 | Voltage-dependent K(+) channels in pancreatic beta cells: role, regulation and potential as therapeutic targets. | Diabetologia | 2003 | https://conductscience.org/library/papers/12830383 | PubMed |
| 10,482,920 | Alterations of insulin secretion from mouse islets treated with sulphonylureas: perturbations of Ca2+ regulation prevail over changes in insulin content. | Br J Pharmacol | 1999 | https://conductscience.org/library/papers/10482920 | PubMed |
| 9,022,074 | Direct glucocorticoid inhibition of insulin secretion. An in vitro study of dexamethasone effects in mouse islets. | J Clin Invest | 1997 | https://conductscience.org/library/papers/9022074 | PubMed |
| 8,789,393 | Increase in insulin release from rat pancreatic islets by quinolone antibiotics. | Br J Pharmacol | 1996 | https://conductscience.org/library/papers/8789393 | PubMed |
| 8,405,748 | Different effects of glucose and glyburide on insulin secretion in rat pancreatic islets pre-exposed to interleukin-1 beta. Possible involvement of K+ and Ca2+ channels. | Diabetologia | 1993 | https://conductscience.org/library/papers/8405748 | PubMed |
| 8,363,584 | Substrate-dependent changes in mitochondrial function, intracellular free calcium concentration and membrane channels in pancreatic beta-cells. | Biochem J | 1993 | https://conductscience.org/library/papers/8363584 | PubMed |
| 8,243,870 | Vasoactive intestinal polypeptide-augmented insulin release: actions on ionic fluxes and electrical activity of mouse islets. | Diabetologia | 1993 | https://conductscience.org/library/papers/8243870 | PubMed |
| 8,110,181 | Two sites of glucose control of insulin release with distinct dependence on the energy state in pancreatic B-cells. | Biochem J | 1994 | https://conductscience.org/library/papers/8110181 | PubMed |
| 7,560,065 | Possible links between glucose-induced changes in the energy state of pancreatic B cells and insulin release. Unmasking by decreasing a stable pool of adenine nucleotides in mouse islets. | J Clin Invest | 1995 | https://conductscience.org/library/papers/7560065 | PubMed |
| 7,031,228 | Regulation of 86Rb outflow from pancreatic islets: the dual effect of nutrient secretagogues. | J Physiol | 1981 | https://conductscience.org/library/papers/7031228 | PubMed |
| 7,027,175 | The stimulus-secretion coupling of amino acid-induced insulin release. IV. Ionic response to L-Leucine and L-Glutamine. | Pflugers Arch | 1981 | https://conductscience.org/library/papers/7027175 | PubMed |
| 7,021,594 | Fluctuations of calcium, phosphorus, sodium, potassium, and chlorine in single alpha and beta cells during glucose perifusion of rat islets. | J Clin Invest | 1981 | https://conductscience.org/library/papers/7021594 | PubMed |
| 7,009,716 | Regulation of 86Rb+ outflow from pancreatic islets III. Possible significance of ATP. | J Endocrinol Invest | 1980 | https://conductscience.org/library/papers/7009716 | PubMed |
| 6,993,267 | Influence of the murine diabetes gene on rubidium ion efflux from perifused islets. | Diabetologia | 1980 | https://conductscience.org/library/papers/6993267 | PubMed |
| 6,988,275 | Tolbutamide stimulation and inhibition of insulin release: studies of the underlying ionic mechanisms in isolated rat islets. | Diabetologia | 1980 | https://conductscience.org/library/papers/6988275 | PubMed |
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- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
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- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
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- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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