Paper · 3526554

A yeast gene that is essential for release from glucose repression encodes a protein kinase.

Celenza JL and 1 other. Science, 1986. Cited by 592 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. A yeast gene that is essential for release from glucose repression encodes a protein kinase. Papers, record 3526554. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/3526554

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: A yeast gene that is essential for release from glucose repression encodes a protein kinase.; PubMed
Journal: Science; PubMed
Year: 1986; PubMed
Publication types: Journal Article, Research Support, Non-U.S. Gov't, Research Support, U.S. Gov't, P.H.S.; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/3526554/; PubMed
DOI: https://doi.org/10.1126/science.3526554; PubMed
The journal: https://conductscience.org/library/journal-index/0404511; PubMed
Citations, by iCite: 592; iCite
Relative citation ratio, by iCite: 11.3; iCite
Revised in PubMed: 2019-06-18; PubMed

Authors2

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename	Kind	ORCID iD
1	Celenza JL	J L
2	Carlson M	M

Source PubMed

Topics8

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Glucose	Major topic
Protein Kinases	Major topic
Saccharomyces cerevisiae	Major topic
Amino Acid Sequence
Base Sequence
Enzyme Repression
Genes
Phosphorylation

Source PubMed

Grants2

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes it	Agency	Country	NIH project	Stated by	Source
GM34095	NIGMS NIH HHS	United States	R01GM034095	PubMed and NIH	NIH RePORTER
			R37GM034095	NIH	NIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References0

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

None in this release.

Cited by283

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 592 citations.

PMID	Title	Journal	Year	In Papers	Source
42,934,234	The AMPK signaling pathway in cell metabolism and human diseases.	Signal Transduct Target Ther	2026	https://conductscience.org/library/papers/42934234	PubMed
38,448,813	Deciphering the regulatory role of PheSnRK genes in Moso bamboo: insights into hormonal, energy, and stress responses.	BMC Genomics	2024	https://conductscience.org/library/papers/38448813	PubMed
37,758,520	Nutrient-dependent signaling pathways that control autophagy in yeast.	FEBS Lett	2024	https://conductscience.org/library/papers/37758520	PubMed
37,450,576	Sucrose non-fermenting protein kinase gene UvSnf1 is required for virulence in Ustilaginoidea virens.	Virulence	2023	https://conductscience.org/library/papers/37450576	PubMed
37,176,842	Comprehensive Genomic Analysis of SnRK in Rosaceae and Expression Analysis of RoSnRK2 in Response to Abiotic Stress in Rubus occidentalis.	Plants (Basel)	2023	https://conductscience.org/library/papers/37176842	PubMed
36,745,535	Convergent and divergent roles of the glucose-responsive kinase SNF4 in Candida tropicalis.	Virulence	2023	https://conductscience.org/library/papers/36745535	PubMed
36,383,046	AMP-activated protein kinase - a journey from 1 to 100 downstream targets.	Biochem J	2022	https://conductscience.org/library/papers/36383046	PubMed
36,316,383	New insights into activation and function of the AMPK.	Nat Rev Mol Cell Biol	2023	https://conductscience.org/library/papers/36316383	PubMed
35,406,955	Genome-Wide Identification, Expression Pattern and Sequence Variation Analysis of SnRK Family Genes in Barley.	Plants (Basel)	2022	https://conductscience.org/library/papers/35406955	PubMed
35,238,938	Modelling of glucose repression signalling in yeast Saccharomyces cerevisiae.	FEMS Yeast Res	2022	https://conductscience.org/library/papers/35238938	PubMed

1 to 10 of 283

See all 283 cited by

Genes1

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

Gene	NCBI Gene ID	Linked by	In Genes
SLK	9,748	PubTator's text mining	https://conductscience.org/library/genes/9748

Source NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data1

The data bank entries PubMed's record names. Trial numbers are under Trials.

Data bank	Accession
GENBANK	M13971

Source GENBANK

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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