Paper · 36096063

The role of dietary factors in nonalcoholic fatty liver disease to hepatocellular carcinoma progression: A systematic review.

Zheng J and 10 others. Clin Nutr, 2022. Cited by 41 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. The role of dietary factors in nonalcoholic fatty liver disease to hepatocellular carcinoma progression: A systematic review. Papers, record 36096063. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/36096063

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: The role of dietary factors in nonalcoholic fatty liver disease to hepatocellular carcinoma progression: A systematic review.; PubMed
Journal: Clin Nutr; PubMed
Year: 2022; PubMed
Publication types: Journal Article, Systematic Review, Research Support, Non-U.S. Gov't; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/36096063/; PubMed
DOI: https://doi.org/10.1016/j.clnu.2022.08.018; PubMed
The journal: https://conductscience.org/library/journal-index/8309603; PubMed
Citations, by iCite: 41; iCite
Relative citation ratio, by iCite: 3.41; iCite
Revised in PubMed: 2026-03-07; PubMed

Authors11

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename
1	Zheng J	Jiali
2	Zhao L	Longgang
3	Dong J	Jingwen
4	Chen H	Huiyi
5	Li D	Donghui
6	Zhang X	Xuehong
7	Hassan MM	Manal M
8	Steck SE	Susan E
9	Li X	Xiaoguang
10	Xiang YB	Yong-Bing

1 to 10 of 11Source PubMed

See all 11 authors

Topics10

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Carcinoma, Hepatocellular	Major topic
Liver Neoplasms	Major topic
Non-alcoholic Fatty Liver Disease	Major topic
Antioxidants
Coffee
Disease Progression
Humans
Inflammation
Liver Cirrhosis
Risk Factors

Source PubMed

Grants1

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes it	Agency	Country	NIH project	Stated by
K07 CA106458	NCI NIH HHS	United States	K07CA106458	PubMed

Source NIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections1

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

Kind	PMID	Title	In Papers
Comment in	36,925,437	Letter to the editor：The role of dietary factors in nonalcoholic fatty liver disease to hepatocellular carcinoma progression: A systematic review.	https://conductscience.org/library/papers/36925437

Source PubMed

References0

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

None in this release.

Cited by24

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 41 citations.

PMID	Title	Journal	Year	In Papers	Source
42,856,957	The changing etiology of liver cancer: A comparative study of global and Chinese burden trends from hepatitis dominance to metabolic risks (1990-2021) and forecasts to 2035.	Liver Res	2026	https://conductscience.org/library/papers/42856957	PubMed
42,581,279	Prophylactic Effects of Dietary Patterns and Indexes, Nutrition and Lifestyle on Hepatocellular Carcinoma.	Ann Surg Oncol	2026	https://conductscience.org/library/papers/42581279	PubMed
42,453,381	Primary Liver Cancer Trends Worldwide and in China: Analysis of GLOBOCAN 2022 Data and Disease Management Implications.	Portal Hypertens Cirrhosis	2026	https://conductscience.org/library/papers/42453381	PubMed
41,908,244	ncRNA-Mediated Upregulation of AURKA Promotes Hepatocellular Carcinoma Progression and Alters the Immune Microenvironment.	Bioinform Biol Insights	2026	https://conductscience.org/library/papers/41908244	PubMed
41,860,879	The association between dietary inflammatory index and non-alcoholic fatty liver disease: A systematic review and meta-analysis.	PLoS One	2026	https://conductscience.org/library/papers/41860879	PubMed
41,667,791	Insulin resistance: mechanisms and therapeutic interventions.	Mol Biomed	2026	https://conductscience.org/library/papers/41667791	PubMed
41,171,441	Chemoprevention of hepatocellular carcinoma.	Hepatol Commun	2025	https://conductscience.org/library/papers/41171441	PubMed
41,146,297	Glucose metabolic dysfunction surpasses dyslipidemia in driving hepatic steatosis and fibrosis: evidence from a nationally representative population.	Diabetol Metab Syndr	2025	https://conductscience.org/library/papers/41146297	PubMed
40,852,740	Coffee and the Risk of Hepatocellular Carcinoma: A Systematic Review and Meta-Analysis of Mendelian Randomization Studies.	Gut Liver	2026	https://conductscience.org/library/papers/40852740	PubMed
40,613,843	Genetic evidence for causal links between diet, gut microbiota, and hepatobiliary cancer: a Mendelian randomization study.	AMB Express	2025	https://conductscience.org/library/papers/40613843	PubMed

1 to 10 of 24

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Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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