Paper · 38705759
Lipid-associated macrophages between aggravation and alleviation of metabolic diseases.
Xu R and 6 others. Trends Endocrinol Metab, 2024. Cited by 89 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Lipid-associated macrophages between aggravation and alleviation of metabolic diseases. Papers, record 38705759. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/38705759
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
Topics6
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Macrophages | Major topic |
| Metabolic Diseases | Major topic |
| Humans | |
| Animals | |
| Lipid Metabolism | |
| Adipose Tissue |
Source PubMed
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References0
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Cited by67
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 89 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 42,941,919 | Integrated Transcriptomic and Single-Cell Analysis Reveals Shared Macrophage-Associated Inflammatory Signatures in MASLD/NAFLD and Rheumatoid Arthritis. | Int J Mol Sci | 2026 | https://conductscience.org/library/papers/42941919 | PubMed |
| 42,935,806 | Obesity-driven inflammation and immune dysregulation in cancer development and progression. | Front Oncol | 2026 | https://conductscience.org/library/papers/42935806 | PubMed |
| 42,911,976 | Multi-omics analysis of high-grade serous ovarian cancers reveals heterogeneity and distinct immunological patterns based upon clinico-genomic status. | PLoS Genet | 2026 | https://conductscience.org/library/papers/42911976 | PubMed |
| 42,894,247 | Cell-resolved genetic risk reveals hyperandrogenism-associated immune and metabolic rewiring in the PMOS follicular microenvironment. | Genome Med | 2026 | https://conductscience.org/library/papers/42894247 | PubMed |
| 42,751,295 | Lipid-Associated Macrophages in Obesity: Immunometabolic Programs and Emerging Therapeutic Implications-A Narrative Review. | Diabetes Metab Syndr Obes | 2026 | https://conductscience.org/library/papers/42751295 | PubMed |
| 42,729,505 | Spatial dynamics of cancer-associated fibroblasts links fibroblastic differentiation to immune exclusion and tumor progression. | Front Immunol | 2026 | https://conductscience.org/library/papers/42729505 | PubMed |
| 42,694,185 | Injured proximal tubule-driven paracrine signaling promotes lipid-associated macrophage expansion and chondroitin sulfate biosynthesis via PPARG/TCF12 in diabetic kidney disease. | Front Immunol | 2026 | https://conductscience.org/library/papers/42694185 | PubMed |
| 42,688,372 | Recruited Monocyte-Derived Macrophages Drive T Cell Inflammation in Immune Checkpoint Inhibitor-Mediated Pneumonitis. | Cancer Commun (Lond) | 2026 | https://conductscience.org/library/papers/42688372 | PubMed |
| 42,666,651 | Spatial immune niche remodeling of the neutrophil-macrophage axis inchronic liver disease. | Front Cell Dev Biol | 2026 | https://conductscience.org/library/papers/42666651 | PubMed |
| 42,660,905 | Dynamic remodeling of the pancreas immune landscape in obesity. | Nat Commun | 2026 | https://conductscience.org/library/papers/42660905 | PubMed |
1 to 10 of 67
See all 67 cited byGenes0
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None in this release.
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None in this release.
Data0
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Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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