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Paper · 38710951

Cited by45

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 64 citations.

PMIDTitleJournalYearIn PapersSource
42,881,857
Molecularly Guided Therapy and the Tumor Microenvironment in Advanced Cholangiocarcinoma: Current Evidence and Emerging Strategies Targeting FGFR2, HER2, and IDH1 with Pemigatinib, Zanidatamab, and Ivosidenib.
J Clin Med2026https://conductscience.org/library/papers/42881857PubMed
42,618,642Exploring the landscape of targetable alterations in patients with glioblastoma.Nat Rev Clin Oncol2026https://conductscience.org/library/papers/42618642PubMed
42,498,744Chinese expert consensus on precision testing and molecular diagnosis of pancreatic cancer (2025).Br J Cancer2026https://conductscience.org/library/papers/42498744PubMed
42,286,159Targetable alterations and personalized treatment in ameloblastoma: results from a prospective observational precision oncology study.NPJ Precis Oncol2026https://conductscience.org/library/papers/42286159PubMed
42,223,137Pemigatinib for Unresectable or Metastatic Cholangiocarcinoma With Fibroblast Growth Factor Receptor-2 Rearrangement: Results From the Phase III FIGHT-302 Trial.J Clin Oncol2026https://conductscience.org/library/papers/42223137PubMed
42,192,928Understanding the Promise and Challenges of Tumor-Agnostic Therapy: Could One Size Really Fit All?Cancers (Basel)2026https://conductscience.org/library/papers/42192928PubMed
42,058,998FGFR alterations and potential role of FGFR inhibitors in pediatric brain tumors.Neurooncol Adv2026https://conductscience.org/library/papers/42058998PubMed
41,874,881Recent Advances in Targeted Therapies for Adult Gliomas.Curr Neurol Neurosci Rep2026https://conductscience.org/library/papers/41874881PubMed
41,849,383The conserved fibroblast growth factor receptor-based signaling is required for dendrite regeneration.Proc Natl Acad Sci U S A2026https://conductscience.org/library/papers/41849383PubMed
41,832,306FGF family in health and disease.Mol Biomed2026https://conductscience.org/library/papers/41832306PubMed
41,793,580Molecular Pathogenesis and Targeted Therapies for Myeloproliferative Hypereosinophilic Neoplasms.Curr Hematol Malig Rep2026https://conductscience.org/library/papers/41793580PubMed
41,682,001FGFR2-Rearranged Biliary Tract Cancer: Biology, Resistance Mechanisms, and Emerging Therapeutic Strategies.Cancers (Basel)2026https://conductscience.org/library/papers/41682001PubMed
41,632,446Recurrent Resistance Mutations to Lirafugratinib Inform Treatment Sequencing in FGFR2-Driven Tumors.Clin Cancer Res2026https://conductscience.org/library/papers/41632446PubMed
41,586,981SREBP2-RAB11A-ZDHHC20 axis orchestrates FGFR3 palmitoylation and membrane retention to drive bladder cancer progression.Cell Oncol (Dordr)2026https://conductscience.org/library/papers/41586981PubMed
41,514,604FGFR Aberrations in Solid Tumors: Mechanistic Insights and Clinical Translation of Targeted Therapies.Cancers (Basel)2025https://conductscience.org/library/papers/41514604PubMed
41,492,079The actionable transcriptome: a framework for incorporating RNA sequencing into precision oncology.Nat Rev Clin Oncol2026https://conductscience.org/library/papers/41492079PubMed
41,460,200Erdafitinib in Patients with FGFR-Altered Advanced or Metastatic Cholangiocarcinoma.Clin Cancer Res2026https://conductscience.org/library/papers/41460200PubMed
41,430,037FGF1-FGFR2 axis regulated by nuclear receptor RORγ represents an effective strategy in intrahepatic cholangiocarcinoma.Cell Death Discov2025https://conductscience.org/library/papers/41430037PubMed
41,374,970KRAS-Wild Pancreatic Cancer-More Targets than Treatment Possibilities?Cancers (Basel)2025https://conductscience.org/library/papers/41374970PubMed
41,361,008Saturation mutagenesis identifies activating and resistance-inducing FGFR kinase domain mutations.Nat Genet2026https://conductscience.org/library/papers/41361008PubMed

papers-2026-11-12-0.1.0

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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