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Paper · 38967973

Primary Distal Renal Tubular Acidosis: Toward an Optimal Correction of Metabolic Acidosis.

Medina E and 2 others. Clin J Am Soc Nephrol, 2024. Cited by 7 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Primary Distal Renal Tubular Acidosis: Toward an Optimal Correction of Metabolic Acidosis. Papers, record 38967973. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/38967973

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Primary Distal Renal Tubular Acidosis: Toward an Optimal Correction of Metabolic Acidosis.
PubMed
Journal
Clin J Am Soc Nephrol
PubMed
Year
2024
PubMed
Publication types
Journal Article, Review
PubMed
Citations, by iCite
7
iCite
Relative citation ratio, by iCite
1.72, provisional
iCite
Revised in PubMed
2026-06-29
PubMed

Authors3

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Source PubMed

Topics7

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Acidosis, Renal TubularMajor topic
Acid-Base EquilibriumMajor topic
AcidosisMajor topic
Humans
Bicarbonates
Treatment Outcome
Nephrocalcinosis

Source PubMed

Grants1

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes itAgencyCountryNIH projectStated by
R21 AI166940NIAID NIH HHSUnited StatesR21AI166940PubMed

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References111

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
129,311,217The Urine Anion Gap in Context.Clin J Am Soc Nephrol2018https://conductscience.org/library/papers/29311217PubMed
232,712,761Efficacy and safety of an innovative prolonged-release combination drug in patients with distal renal tubular acidosis: an open-label comparative trial versus standard of care treatments.Pediatr Nephrol2021https://conductscience.org/library/papers/32712761PubMed
330,139,463Incomplete Distal Renal Tubular Acidosis and Kidney Stones.Adv Chronic Kidney Dis2018https://conductscience.org/library/papers/30139463PubMed
428,599,903Metabolic Acidosis or Respiratory Alkalosis? Evaluation of a Low Plasma Bicarbonate Using the Urine Anion Gap.Am J Kidney Dis2017https://conductscience.org/library/papers/28599903PubMed
57,036,725Hyperkalemic hyperchloremic metabolic acidosis in sickle cell hemoglobinopathies.Am J Med1982https://conductscience.org/library/papers/7036725PubMed
619,277,700Regulated acid-base transport in the collecting duct.Pflugers Arch2009https://conductscience.org/library/papers/19277700PubMed
720,598,674Establishment of pediatric reference intervals on a large cohort of healthy children.Clin Chim Acta2010https://conductscience.org/library/papers/20598674PubMed
81,450,372On the mechanism of impaired distal acidification in hyperkalemic renal tubular acidosis: evaluation with amiloride and bumetanide.J Am Soc Nephrol1992https://conductscience.org/library/papers/1450372PubMed
99,497,368Autosomal dominant distal renal tubular acidosis is associated in three families with heterozygosity for the R589H mutation in the AE1 (band 3) Cl-/HCO3- exchanger.J Biol Chem1998https://conductscience.org/library/papers/9497368PubMed
108,301,923Effect of metabolic acidosis on the expression of insulin-like growth factor and growth hormone receptor.Kidney Int1993https://conductscience.org/library/papers/8301923PubMed

Cited by6

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 7 citations.

PMIDTitleJournalYearIn PapersSource
42,862,495Early-onset autosomal dominant distal renal tubular acidosis caused by a de novo SLC4A1 R589C variant with growth failure and rickets.CEN Case Rep2026https://conductscience.org/library/papers/42862495PubMed
42,694,869Sjögren's Disease-Associated Renal Tubular Acidosis: An Integrated Immuno-Tubular Model of Tubular Dysfunction.Int J Med Sci2026https://conductscience.org/library/papers/42694869PubMed
41,615,714Nephrocalcinosis: unveiling renal tubulopathies in the genomic era.J Bras Nefrol2026https://conductscience.org/library/papers/41615714PubMed
40,775,604Identification of novel pathogenic mutations in ATP6V0A4 associated with distal renal tubular acidosis and analysis of wild-type expression in glomerular disease.Ren Fail2025https://conductscience.org/library/papers/40775604PubMed
40,299,568A gain-of-function mutation in ATP6V0A4 drives primary distal renal tubular alkalosis with enhanced V-ATPase activity.J Clin Invest2025https://conductscience.org/library/papers/40299568PubMed
39,835,572Potassium-Rich, Gluten-Free Diets for Patients with Sjögren's Syndrome: A Mini-ReviewEndocr Metab Immune Disord Drug Targets2026https://conductscience.org/library/papers/39835572PubMed

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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