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Paper · 39015481

Dysfunctional mitochondria elicit bioenergetic decline in the aged heart.

Mone P and 7 others. J Cardiovasc Aging, 2024. Cited by 15 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Dysfunctional mitochondria elicit bioenergetic decline in the aged heart. Papers, record 39015481. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/39015481

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Dysfunctional mitochondria elicit bioenergetic decline in the aged heart.
PubMed
Journal
J Cardiovasc Aging
PubMed
Year
2024
PubMed
Publication types
Journal Article
PubMed
Citations, by iCite
15
iCite
Relative citation ratio, by iCite
2.47, provisional
iCite
Revised in PubMed
2024-07-19
PubMed

Authors8

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Mone PPasquale
2Agyapong EDEsther Densu
3Morciano GGiampaolo
4Jankauskas SSStanislovas S
5De Luca AAntonio
6Varzideh FFahimeh
7Pinton PPaolo
8Santulli GGaetanohttps://orcid.org/0000-0001-7231-375X

Source PubMed

Topics0

NLM's MeSH indexing of the paper, its major topics first.

None in this release.

Grants8

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes itAgencyCountryNIH projectStated bySource
R01 HL164772NHLBI NIH HHSUnited StatesR01HL164772PubMed and NIHNIH RePORTER
R01 DK123259NIDDK NIH HHSUnited StatesR01DK123259PubMed and NIHNIH RePORTER
R01 DK033823NIDDK NIH HHSUnited StatesR01DK033823PubMed and NIHNIH RePORTER
R01 HL159062NHLBI NIH HHSUnited StatesR01HL159062PubMed and NIHNIH RePORTER
UM1 TR004400NCATS NIH HHSUnited StatesUM1TR004400PubMed and NIHNIH RePORTER
UL1 TR002556NCATS NIH HHSUnited StatesUL1TR002556PubMed and NIHNIH RePORTER
R01 HL146691NHLBI NIH HHSUnited StatesR01HL146691PubMed and NIHNIH RePORTER
T32 HL144456NHLBI NIH HHSUnited StatesT32HL144456PubMed and NIHNIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References199

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
115,879,174Extension of murine life span by overexpression of catalase targeted to mitochondria.Science2005https://conductscience.org/library/papers/15879174PubMed
225,609,139Mitophagy and heart failure.J Mol Med (Berl)2015https://conductscience.org/library/papers/25609139PubMed
334,369,184Effects of Aging on Cardiac Oxidative Stress and Transcriptional Changes in Pathways of Reactive Oxygen Species Generation and Clearance.J Am Heart Assoc2021https://conductscience.org/library/papers/34369184PubMed
422,406,748Down-regulation of OPA1 alters mouse mitochondrial morphology, PTP function, and cardiac adaptation to pressure overload.Cardiovasc Res2012https://conductscience.org/library/papers/22406748PubMed
538,066,057Downregulation of mitochondrial metabolism is a driver for fast skeletal muscle loss during mouse aging.Commun Biol2023https://conductscience.org/library/papers/38066057PubMed
631,115,493Mitochondrial electron transport chain, ROS generation and uncoupling (Review).Int J Mol Med2019https://conductscience.org/library/papers/31115493PubMed
729,054,534Mechanisms driving the ageing heart.Exp Gerontol2018https://conductscience.org/library/papers/29054534PubMed
826,370,078Resistance of Dynamin-related Protein 1 Oligomers to Disassembly Impairs Mitophagy, Resulting in Myocardial Inflammation and Heart Failure.J Biol Chem2015https://conductscience.org/library/papers/26370078PubMed
920,576,649Mechanisms of vascular aging: new perspectives.J Gerontol A Biol Sci Med Sci2010https://conductscience.org/library/papers/20576649PubMed
1032,714,204Lifestyle, Oxidative Stress, and Antioxidants: Back and Forth in the Pathophysiology of Chronic Diseases.Front Physiol2020https://conductscience.org/library/papers/32714204PubMed

1 to 10 of 199

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Cited by13

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 15 citations.

PMIDTitleJournalYearIn PapersSource
42,928,775Targeting Metabolic Drivers of Cardiovascular Disease: Insights for Therapeutic Innovation.Drug Des Devel Ther2026https://conductscience.org/library/papers/42928775PubMed
42,662,706Understanding cardiovascular aging as a disorder of mitochondrial network.J Cardiovasc Aging2026https://conductscience.org/library/papers/42662706PubMed
42,010,677Harnessing the novel safeguarding role of Selenoprotein T in age-related myocardial left and right decline through the ferroptosis-mitochondrial axis.J Transl Med2026https://conductscience.org/library/papers/42010677PubMed
41,654,866The importance of mitochondria and mitochondrial calcium signaling in health and disease: an updated outlook on inflammation.J Transl Med2026https://conductscience.org/library/papers/41654866PubMed
41,361,441Interplay between frailty and cardiometabolic disorders: from pathophysiology to clinical implications.Cardiovasc Diabetol2025https://conductscience.org/library/papers/41361441PubMed
41,301,698Mitochondrial Dynamics in Aging Heart.Biomedicines2025https://conductscience.org/library/papers/41301698PubMed
41,180,389Logistic Regression and Machine Learning Algorithms for the Risk Prediction of Perioperative Adverse Cardiovascular Events in Elderly Patients.Aging Med (Milton)2025https://conductscience.org/library/papers/41180389PubMed
41,155,179Mechanisms of Mitochondrial Impairment by SARS-CoV-2 Proteins: A Nexus of Pathogenesis with Significant Biochemical and Clinical Implications.Int J Mol Sci2025https://conductscience.org/library/papers/41155179PubMed
41,146,020Cellular and molecular mechanisms underlying cardiovascular aging.Cell Mol Biol Lett2025https://conductscience.org/library/papers/41146020PubMed
40,953,136Intensity, Duration, and Context Dependency of the Responses to Nutrient Surplus and Deprivation Signaling in the Heart: Insights Into the Complexities of Cardioprotection.Circulation2025https://conductscience.org/library/papers/40953136PubMed

1 to 10 of 13

See all 13 cited by

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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