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Paper · 39397601

Vitamin A Deficiency Disturbs Ret Expression and Induces Urinary Tract Developmental Abnormalities in Mice.

Yu M and 7 others. Am J Nephrol, 2025. Cited by 5 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Vitamin A Deficiency Disturbs Ret Expression and Induces Urinary Tract Developmental Abnormalities in Mice. Papers, record 39397601. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/39397601

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Vitamin A Deficiency Disturbs Ret Expression and Induces Urinary Tract Developmental Abnormalities in Mice.
PubMed
Journal
Am J Nephrol
PubMed
Year
2025
PubMed
Publication types
Journal Article
PubMed
Citations, by iCite
5
iCite
Relative citation ratio, by iCite
1.52, provisional
iCite
Revised in PubMed
2026-05-13
PubMed

Authors8

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Yu MMinghui
2Ju HHaixin
3Ye NNingli
4Chen JJing
5Sun LLei
6Wu XXiaohui
7Xu HHong
8Shen QQian

Source PubMed

Topics13

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Vitamin A DeficiencyMajor topic
Vitamin AMajor topic
Proto-Oncogene Proteins c-retMajor topic
Urogenital AbnormalitiesMajor topic
Urinary TractMajor topic
Animals
Female
Pregnancy
Mice
Disease Models, Animal
Male
Mice, Inbred C57BL
Vesico-Ureteral Reflux

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References30

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
111,480,433Fetal toxic effects and angiotensin-II-receptor antagonists.Lancet2001https://conductscience.org/library/papers/11480433PubMed
225,270,717Copy-number variation associated with congenital anomalies of the kidney and urinary tract.Pediatr Nephrol2015https://conductscience.org/library/papers/25270717PubMed
317,093,988Effects of maternal vitamin A status on kidney development: a pilot study.Pediatr Nephrol2007https://conductscience.org/library/papers/17093988PubMed
424,398,540Single-gene causes of congenital anomalies of the kidney and urinary tract (CAKUT) in humans.Pediatr Nephrol2014https://conductscience.org/library/papers/24398540PubMed
512,195,422Distal ureter morphogenesis depends on epithelial cell remodeling mediated by vitamin A and Ret.Nat Genet2002https://conductscience.org/library/papers/12195422PubMed
612,373,613Determinants of weight and length of Indonesian neonates.Eur J Clin Nutr2002https://conductscience.org/library/papers/12373613PubMed
738,226,115Vitamin A-Mediated Birth Defects: A Narrative Review.Cureus2023https://conductscience.org/library/papers/38226115PubMed
824,744,441Prenatal risk factors for childhood CKD.J Am Soc Nephrol2014https://conductscience.org/library/papers/24744441PubMed
912,401,878Gene expression regulation by retinoic acid.J Lipid Res2002https://conductscience.org/library/papers/12401878PubMed
1031,506,986Detection of copy number disorders associated with congenital anomalies of the kidney and urinary tract in fetuses via single nucleotide polymorphism arrays.J Clin Lab Anal2020https://conductscience.org/library/papers/31506986PubMed

1 to 10 of 30

See all 30 references

Cited by4

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 5 citations.

PMIDTitleJournalYearIn PapersSource
41,977,230Vitamin D Deficiency Activates Gdnf-Ret-pErk1/2 Signal and Induces Kidney Malformations in Mice.Int J Mol Sci2026https://conductscience.org/library/papers/41977230PubMed
41,304,543Maternal Exposure to 2,4-Di-tert-butylphenol During Pregnancy in a Mouse Model Leads to Abnormal Development of the Urinary System in Offspring.Toxics2025https://conductscience.org/library/papers/41304543PubMed
40,621,246Vitamin A Deficiency in Children With Neurodevelopmental Disorders: Case Reports of Ocular and Urinary Tract Complications in Two Patients.Cureus2025https://conductscience.org/library/papers/40621246PubMed
40,164,775Ureter development and associated congenital anomalies.Nat Rev Nephrol2025https://conductscience.org/library/papers/40164775PubMed

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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