Paper · 39521920
Neurexin facilitates glycosylation of Dystroglycan to sustain muscle architecture and function in Drosophila.
Zhao Y and 10 others. Commun Biol, 2024. Cited by 3 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Neurexin facilitates glycosylation of Dystroglycan to sustain muscle architecture and function in Drosophila. Papers, record 39521920. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/39521920
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Neurexin facilitates glycosylation of Dystroglycan to sustain muscle architecture and function in Drosophila.
- PubMed
Topics9
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Dystroglycans | Major topic |
| Drosophila Proteins | Major topic |
| Drosophila melanogaster | Major topic |
| Animals | |
| Glycosylation | |
| Muscles | |
| Drosophila | |
| Mutation | |
| Cell Adhesion Molecules, Neuronal |
Source PubMed
Grants3
Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.
| Grant, as the record writes it | Agency | Country | NIH project | Stated by |
|---|---|---|---|---|
| 32271013 | National Natural Science Foundation of China (National Science Foundation of China) | PubMed | ||
| 31771592 | National Natural Science Foundation of China (National Science Foundation of China) | PubMed | ||
| 32171149 | National Natural Science Foundation of China (National Science Foundation of China) | PubMed |
Source PubMed
Trials0
The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.
None in this release.
Comments and corrections0
PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.
None in this release.
References68
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 17,498,701 | Neurexin-1 is required for synapse formation and larvae associative learning in Drosophila. | FEBS Lett | 2007 | https://conductscience.org/library/papers/17498701 | PubMed |
| 2 | 21,452,199 | Conditional knockout of protein O-mannosyltransferase 2 reveals tissue-specific roles of O-mannosyl glycosylation in brain development. | J Comp Neurol | 2011 | https://conductscience.org/library/papers/21452199 | PubMed |
| 3 | 12,441,301 | Dystroglycan is required for polarizing the epithelial cells and the oocyte in Drosophila. | Development | 2003 | https://conductscience.org/library/papers/12441301 | PubMed |
| 4 | 23,352,167 | Neurexin regulates visual function via mediating retinoid transport to promote rhodopsin maturation. | Neuron | 2013 | https://conductscience.org/library/papers/23352167 | PubMed |
| 5 | 23,107,381 | Dg-Dys-Syn1 signaling in Drosophila regulates the microRNA profile. | BMC Cell Biol | 2012 | https://conductscience.org/library/papers/23107381 | PubMed |
| 6 | 28,810,660 | Recent advancements in understanding mammalian O-mannosylation. | Glycobiology | 2017 | https://conductscience.org/library/papers/28810660 | PubMed |
| 7 | 17,881,734 | Dystroglycan and protein O-mannosyltransferases 1 and 2 are required to maintain integrity of Drosophila larval muscles. | Mol Biol Cell | 2007 | https://conductscience.org/library/papers/17881734 | PubMed |
| 8 | 20,547,130 | Drosophila neuroligin 1 promotes growth and postsynaptic differentiation at glutamatergic neuromuscular junctions. | Neuron | 2010 | https://conductscience.org/library/papers/20547130 | PubMed |
| 9 | 19,969,597 | Drosophila Dystroglycan is a target of O-mannosyltransferase activity of two protein O-mannosyltransferases, Rotated Abdomen and Twisted. | Glycobiology | 2010 | https://conductscience.org/library/papers/19969597 | PubMed |
| 10 | 17,035,546 | Important contribution of alpha-neurexins to Ca2+-triggered exocytosis of secretory granules. | J Neurosci | 2006 | https://conductscience.org/library/papers/17035546 | PubMed |
1 to 10 of 68
See all 68 referencesCited by1
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 3 citations.
| PMID | Title | Journal | Year | In Papers |
|---|---|---|---|---|
| 42,039,277 | Aberrant astroglial Kir4.1 activation in the anterior cingulate cortex disrupts neuronal excitability and social behavior. | Acta Pharm Sin B | 2026 | https://conductscience.org/library/papers/42039277 |
Source PubMed
Genes1
The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.
| Gene | NCBI Gene ID | Linked by | In Genes |
|---|---|---|---|
| POMT1 | 10,585 | PubTator's text mining | https://conductscience.org/library/genes/10585 |
Source NCBI Gene
Drugs0
The drugs whose trials, in the Pipelines release, list the paper.
None in this release.
Data0
The data bank entries PubMed's record names. Trial numbers are under Trials.
None in this release.
Data dates
The date of each source this release read.
- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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