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Paper · 39697181

Cyclic di-AMP regulates genome stability and drug resistance in Mycobacterium through RecA-dependent and RecA-independent recombination.

Mudgal S and 8 others. PNAS Nexus, 2024. Cited by 5 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Cyclic di-AMP regulates genome stability and drug resistance in Mycobacterium through RecA-dependent and RecA-independent recombination. Papers, record 39697181. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/39697181

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Cyclic di-AMP regulates genome stability and drug resistance in Mycobacterium through RecA-dependent and RecA-independent recombination.
PubMed
Journal
PNAS Nexus
PubMed
Year
2024
PubMed
Publication types
Journal Article
PubMed
Citations, by iCite
5
iCite
Relative citation ratio, by iCite
0.79, provisional
iCite
Revised in PubMed
2025-01-04
PubMed

Authors9

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Mudgal SSudhanshuhttps://orcid.org/0009-0009-4349-9781
2Goyal NNishahttps://orcid.org/0009-0002-0161-631X
3Kasi MManikandanhttps://orcid.org/0000-0003-1567-1264
4Saginela RRahulhttps://orcid.org/0009-0000-7422-5366
5Singhal AAnusha
6Nandi SSoumyadeep
7Mahmud AKMFA K M Firojhttps://orcid.org/0009-0007-1494-8344
8Muniyappa KKalappahttps://orcid.org/0000-0002-9192-9194
9Sinha KMKrishna Murarihttps://orcid.org/0000-0002-9420-9432

Source PubMed

Topics0

NLM's MeSH indexing of the paper, its major topics first.

None in this release.

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References76

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
116,713,562A checkpoint protein that scans the chromosome for damage at the start of sporulation in Bacillus subtilis.Cell2006https://conductscience.org/library/papers/16713562PubMed
237,444,780Antimicrobial Resistance: A Growing Serious Threat for Global Public Health.Healthcare (Basel)2023https://conductscience.org/library/papers/37444780PubMed
312,705,867DnaE2 polymerase contributes to in vivo survival and the emergence of drug resistance in Mycobacterium tuberculosis.Cell2003https://conductscience.org/library/papers/12705867PubMed
429,873,124
The second messenger cyclic di-AMP negatively regulates the expression of Mycobacterium smegmatis recA and attenuates DNA strand exchange through binding to the C-terminal motif of mycobacterial RecA proteins.
Mol Microbiol2018https://conductscience.org/library/papers/29873124PubMed
515,620,655The small RNA IstR inhibits synthesis of an SOS-induced toxic peptide.Curr Biol2004https://conductscience.org/library/papers/15620655PubMed
634,715,302Cyclic di-AMP: Small molecule with big roles in bacteria.Microb Pathog2021https://conductscience.org/library/papers/34715302PubMed
726,525,148An Overview of the Molecular Mechanisms of Recombinational DNA Repair.Cold Spring Harb Perspect Biol2015https://conductscience.org/library/papers/26525148PubMed
816,107,138A molecular target for suppression of the evolution of antibiotic resistance: inhibition of the Escherichia coli RecA protein by N(6)-(1-naphthyl)-ADP.J Med Chem2005https://conductscience.org/library/papers/16107138PubMed
917,228,330Motoring along with the bacterial RecA protein.Nat Rev Mol Cell Biol2007https://conductscience.org/library/papers/17228330PubMed
1028,369,307Antimicrobial resistance in Mycobacterium tuberculosis: mechanistic and evolutionary perspectives.FEMS Microbiol Rev2017https://conductscience.org/library/papers/28369307PubMed

1 to 10 of 76

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Cited by3

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 5 citations.

PMIDTitleJournalYearIn PapersSource
42,928,305Rhythmic-phasic c-di-AMP signaling: a new paradigm for bacterial growth synchronization governed by a pseudo-phosphodiesterase and modifiable diadenylate cyclase.Cell Commun Signal2026https://conductscience.org/library/papers/42928305PubMed
41,988,085Structural and biochemical insights into the inhibition of Mycobacterium tuberculosis cyclic dinucleotide phosphodiesterase by a sulfur-modified cyclic dinucleotide analog.RSC Chem Biol2026https://conductscience.org/library/papers/41988085PubMed
41,837,629Mycobacterium tuberculosis as a model system.J Bacteriol2026https://conductscience.org/library/papers/41837629PubMed

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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