ConductScience.org, home
Papers
API referenceFor AI assistants

Paper · 39744429

Tail Fin Regeneration in Zebrafish: The Role of Non-canonical Crosstalk Between STAT3 and Vitamin D Pathway.

Tesoriere A and 7 others. Int J Biol Sci, 2025. Cited by 3 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

Download JSON
Cite this record

ConductScience Library. Tail Fin Regeneration in Zebrafish: The Role of Non-canonical Crosstalk Between STAT3 and Vitamin D Pathway. Papers, record 39744429. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/39744429

On this page

Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Tail Fin Regeneration in Zebrafish: The Role of Non-canonical Crosstalk Between STAT3 and Vitamin D Pathway.
PubMed
Journal
Int J Biol Sci
PubMed
Year
2025
PubMed
Publication types
Journal Article, Research Support, Non-U.S. Gov't
PubMed
Citations, by iCite
3
iCite
Revised in PubMed
2026-07-31
PubMed

Authors8

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Tesoriere AAnnachiara
2Ghirardo RRachele
3Terrin FFrancesca
4Sernesi FFrancesco
5Meneghetti GGiacomo
6Dalla Valle LLuisa
7Dinarello AAlberto
8Argenton FFrancesco

Source PubMed

Topics10

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
ZebrafishMajor topic
STAT3 Transcription FactorMajor topic
RegenerationMajor topic
Animal FinsMajor topic
Vitamin DMajor topic
Zebrafish ProteinsMajor topic
Animals
Signal Transduction
Phosphorylation
Tail

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References66

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
114,673,173Essential role of STAT3 in postnatal survival and growth revealed by mice lacking STAT3 serine 727 phosphorylation.Mol Cell Biol2004https://conductscience.org/library/papers/14673173PubMed
225,015,343Vitamin D promotes vascular regeneration.Circulation2014https://conductscience.org/library/papers/25015343PubMed
324,312,439The MEK-ERK pathway is necessary for serine phosphorylation of mitochondrial STAT3 and Ras-mediated transformation.PLoS One2013https://conductscience.org/library/papers/24312439PubMed
435,670,763IL-10 promotes endothelial progenitor cell infiltration and wound healing via STAT3.FASEB J2022https://conductscience.org/library/papers/35670763PubMed
534,473,253Y705 and S727 are required for the mitochondrial import and transcriptional activities of STAT3, and for regulation of stem cell proliferation.Development2021https://conductscience.org/library/papers/34473253PubMed
622,782,502Molecular mechanisms of vitamin D action.Calcif Tissue Int2013https://conductscience.org/library/papers/22782502PubMed
731,178,754Understanding the Metabolic Profile of Macrophages During the Regenerative Process in Zebrafish.Front Physiol2019https://conductscience.org/library/papers/31178754PubMed
834,137,790Human STAT3 variants underlie autosomal dominant hyper-IgE syndrome by negative dominance.J Exp Med2021https://conductscience.org/library/papers/34137790PubMed
911,322,950Ser727-dependent transcriptional activation by association of p300 with STAT3 upon IL-6 stimulation.FEBS Lett2001https://conductscience.org/library/papers/11322950PubMed
1019,962,667The JAK2 inhibitor AZD1480 potently blocks Stat3 signaling and oncogenesis in solid tumors.Cancer Cell2009https://conductscience.org/library/papers/19962667PubMed

1 to 10 of 66

See all 66 references

Cited by3

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 3 citations.

PMIDTitleJournalYearIn PapersSource
42,275,222Synergistic cooperation between progranulin and Jak2/Stat3 signaling determines definitive myeloid cell fate.Cell Rep2026https://conductscience.org/library/papers/42275222PubMed
42,082,968β-Sitosterol β-D-glucoside (BSSG) triggers intestinal inflammation in zebrafish and mouse models prior to neurodegeneration onset.J Biomed Sci2026https://conductscience.org/library/papers/42082968PubMed
41,997,945Mytho/Phaf1 is required to prevent DNA damage and tissue degeneration in Danio rerio.Cell Death Discov2026https://conductscience.org/library/papers/41997945PubMed

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

For software

Read this paper from software.

The record and each of its lists are JSON, twenty rows a page. No key.

GET https://conductscience.org/api/papers/v1/39744429

GET https://conductscience.org/api/papers/v1/39744429/references?page=2

API referenceFor AI assistants