Paper · 40162097
Absence of T-box transcription factor 21 limits neuromuscular junction recovery after nerve injury in T-bet-knockout mice.
Jablonka-Shariff A and 2 others. Front Cell Dev Biol, 2025. Cited by 0 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Absence of T-box transcription factor 21 limits neuromuscular junction recovery after nerve injury in T-bet-knockout mice. Papers, record 40162097. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/40162097
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Absence of T-box transcription factor 21 limits neuromuscular junction recovery after nerve injury in T-bet-knockout mice.
- PubMed
Topics0
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None in this release.
Grants1
Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.
| Grant, as the record writes it | Agency | Country | NIH project | Stated by |
|---|---|---|---|---|
| T35 HL007815 | NHLBI NIH HHS | United States | T35HL007815 | PubMed |
Source NIH RePORTER
Trials0
The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.
None in this release.
Comments and corrections0
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References55
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 26,279,190 | Macrophage-Induced Blood Vessels Guide Schwann Cell-Mediated Regeneration of Peripheral Nerves. | Cell | 2015 | https://conductscience.org/library/papers/26279190 | PubMed |
| 2 | 24,790,203 | Terminal Schwann cells participate in neuromuscular synapse remodeling during reinnervation following nerve injury. | J Neurosci | 2014 | https://conductscience.org/library/papers/24790203 | PubMed |
| 3 | 21,497,118 | Asymmetric proteasome segregation as a mechanism for unequal partitioning of the transcription factor T-bet during T lymphocyte division. | Immunity | 2011 | https://conductscience.org/library/papers/21497118 | PubMed |
| 4 | 22,573,321 | Live imaging reveals differing roles of macrophages and neutrophils during zebrafish tail fin regeneration. | J Biol Chem | 2012 | https://conductscience.org/library/papers/22573321 | PubMed |
| 5 | 19,805,038 | The transcription factors T-bet and GATA-3 control alternative pathways of T-cell differentiation through a shared set of target genes. | Proc Natl Acad Sci U S A | 2009 | https://conductscience.org/library/papers/19805038 | PubMed |
| 6 | 35,474,470 | Functional adaptation of glial cells at neuromuscular junctions in response to injury. | Glia | 2022 | https://conductscience.org/library/papers/35474470 | PubMed |
| 7 | 29,851,253 | Hypothalamic Sirt1 protects terminal Schwann cells and neuromuscular junctions from age-related morphological changes. | Aging Cell | 2018 | https://conductscience.org/library/papers/29851253 | PubMed |
| 8 | 18,353,892 | Endogenous interferon-gamma is required for efficient skeletal muscle regeneration. | Am J Physiol Cell Physiol | 2008 | https://conductscience.org/library/papers/18353892 | PubMed |
| 9 | 37,355,220 | Neutrophil biology in injuries and diseases of the central and peripheral nervous systems. | Prog Neurobiol | 2023 | https://conductscience.org/library/papers/37355220 | PubMed |
| 10 | 37,554,723 | The current state and future of T-cell exhaustion research. | Oxf Open Immunol | 2023 | https://conductscience.org/library/papers/37554723 | PubMed |
1 to 10 of 55
See all 55 referencesCited by0
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Drugs0
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Data0
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Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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