Paper · 40937495

Retinoic Acid Inhibition Alters Intestinal Composition in Zebrafish: A Non-genetic Model to Study Hirschsprung Disease?

Kakiailatu NJM and 8 others. Neurogastroenterol Motil, 2025. Cited by 0 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Retinoic Acid Inhibition Alters Intestinal Composition in Zebrafish: A Non-genetic Model to Study Hirschsprung Disease? Papers, record 40937495. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/40937495

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: Retinoic Acid Inhibition Alters Intestinal Composition in Zebrafish: A Non-genetic Model to Study Hirschsprung Disease?; PubMed
Journal: Neurogastroenterol Motil; PubMed
Year: 2025; PubMed
Publication types: Journal Article; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/40937495/; PubMed
DOI: https://doi.org/10.1111/nmo.70155; PubMed
PMC: https://pmc.ncbi.nlm.nih.gov/articles/PMC12534589/; PubMed
The journal: https://conductscience.org/library/journal-index/9432572; PubMed
Citations, by iCite: 0; iCite
Revised in PubMed: 2026-09-25; PubMed

Authors9

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename	ORCID iD
1	Kakiailatu NJM	Naomi J M
2	Zhang W	Wei
3	Kuil LE	Laura E
4	Bindels E	Eric
5	Zink JTM	Joke T M
6	Vermeulen M	Michael
7	de Pater E	Emma
8	Melotte V	Veerle	https://orcid.org/0000-0002-9459-123X
9	Alves MM	Maria M

Source PubMed

Topics8

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Hirschsprung Disease	Major topic
Tretinoin	Major topic
Enteric Nervous System	Major topic
Intestines	Major topic
Animals
Zebrafish
Disease Models, Animal
Proto-Oncogene Proteins c-ret

Source PubMed

Grants1

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes it	Agency	Country	NIH project	Stated by
	The Friends of the Sophia Foundation			PubMed

Source PubMed

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References76

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

Order	PMID	Title	Journal	Year	In Papers	Source
1	29,503,736	Control of Innate and Adaptive Lymphocytes by the RAR-Retinoic Acid Axis.	Immune Netw	2018	https://conductscience.org/library/papers/29503736	PubMed
2	32,219,956	Regulation of the intestinal barrier by nutrients: The role of tight junctions.	Anim Sci J	2020	https://conductscience.org/library/papers/32219956	PubMed
3	23,639,815	Enteric nervous system development: migration, differentiation, and disease.	Am J Physiol Gastrointest Liver Physiol	2013	https://conductscience.org/library/papers/23639815	PubMed
4	31,108,231	The Enteric Nervous System for Epithelial Researchers: Basic Anatomy, Techniques, and Interactions With the Epithelium.	Cell Mol Gastroenterol Hepatol	2019	https://conductscience.org/library/papers/31108231	PubMed
5	18,778,953	Retinoic acid-dependent regulation of immune responses by dendritic cells and macrophages.	Semin Immunol	2009	https://conductscience.org/library/papers/18778953	PubMed
6	17,215,310	Specification of epibranchial placodes in zebrafish.	Development	2007	https://conductscience.org/library/papers/17215310	PubMed
7	23,717,535	Retinoic acid upregulates ret and induces chain migration and population expansion in vagal neural crest cells to colonise the embryonic gut.	PLoS One	2013	https://conductscience.org/library/papers/23717535	PubMed
8	33,352,109	Enteroendocrine cells sense bacterial tryptophan catabolites to activate enteric and vagal neuronal pathways.	Cell Host Microbe	2021	https://conductscience.org/library/papers/33352109	PubMed
9	18,402,640	The enteric nervous system: normal functions and enteric neuropathies.	Neurogastroenterol Motil	2008	https://conductscience.org/library/papers/18402640	PubMed
10	31,399,314	Retinoic acid promotes barrier functions in human iPSC-derived intestinal epithelial monolayers.	J Pharmacol Sci	2019	https://conductscience.org/library/papers/31399314	PubMed

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Cited by0

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 0 citations.

None in this release.

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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