Paper · 40948827
Enhancing the sensitivity of lung adenocarcinoma to immune therapeutic agents through SPRED1.
Wu C and 4 others. Transl Lung Cancer Res, 2025. Cited by 0 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Enhancing the sensitivity of lung adenocarcinoma to immune therapeutic agents through SPRED1. Papers, record 40948827. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/40948827
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Enhancing the sensitivity of lung adenocarcinoma to immune therapeutic agents through SPRED1.
- PubMed
Topics0
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None in this release.
Grants0
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Trials0
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None in this release.
Comments and corrections0
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None in this release.
References42
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 18,097,448 | Immunological aspects of cancer chemotherapy. | Nat Rev Immunol | 2008 | https://conductscience.org/library/papers/18097448 | PubMed |
| 2 | 25,682,184 | The prognostic value of PD-L1 expression for non-small cell lung cancer patients: a meta-analysis. | Eur J Surg Oncol | 2015 | https://conductscience.org/library/papers/25682184 | PubMed |
| 3 | 28,223,824 | miR182 activates the Ras-MEK-ERK pathway in human oral cavity squamous cell carcinoma by suppressing RASA1 and SPRED1. | Onco Targets Ther | 2017 | https://conductscience.org/library/papers/28223824 | PubMed |
| 4 | 20,505,728 | The mutation spectrum revealed by paired genome sequences from a lung cancer patient. | Nature | 2010 | https://conductscience.org/library/papers/20505728 | PubMed |
| 5 | 28,684,311 | Hippo effector YAP directly regulates the expression of PD-L1 transcripts in EGFR-TKI-resistant lung adenocarcinoma. | Biochem Biophys Res Commun | 2017 | https://conductscience.org/library/papers/28684311 | PubMed |
| 6 | 16,337,795 | Sprouty proteins: multifaceted negative-feedback regulators of receptor tyrosine kinase signaling. | Trends Cell Biol | 2006 | https://conductscience.org/library/papers/16337795 | PubMed |
| 7 | 30,874,331 | The Sprouty/Spred family as tumor suppressors: Coming of age. | Cancer Sci | 2019 | https://conductscience.org/library/papers/30874331 | PubMed |
| 8 | 36,633,525 | Cancer statistics, 2023. | CA Cancer J Clin | 2023 | https://conductscience.org/library/papers/36633525 | PubMed |
| 9 | 28,102,259 | Elements of cancer immunity and the cancer-immune set point. | Nature | 2017 | https://conductscience.org/library/papers/28102259 | PubMed |
| 10 | 33,734,139 | Changing profiles of cancer burden worldwide and in China: a secondary analysis of the global cancer statistics 2020. | Chin Med J (Engl) | 2021 | https://conductscience.org/library/papers/33734139 | PubMed |
1 to 10 of 42
See all 42 referencesCited by0
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 0 citations.
None in this release.
Genes2
The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.
| Gene | NCBI Gene ID | Linked by | In Genes | Source |
|---|---|---|---|---|
| CD274 | 29,126 | PubTator's text mining | https://conductscience.org/library/genes/29126 | NCBI Gene |
| SPRED1 | 161,742 | PubTator's text mining | https://conductscience.org/library/genes/161742 | NCBI Gene |
Drugs0
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None in this release.
Data0
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None in this release.
Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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