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Paper · 41333154

Alpha-neurexins in health and disease.

Chofflet N and 3 others. Front Mol Neurosci, 2025. Cited by 4 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Alpha-neurexins in health and disease. Papers, record 41333154. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/41333154

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Alpha-neurexins in health and disease.
PubMed
Journal
Front Mol Neurosci
PubMed
Year
2025
PubMed
Publication types
Journal Article, Review
PubMed
Citations, by iCite
4
iCite
Revised in PubMed
2025-12-05
PubMed

Authors4

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Chofflet NNicolas
2Wang MManni
3Chofflet MMathilde
4Takahashi HHideto

Source PubMed

Topics0

NLM's MeSH indexing of the paper, its major topics first.

None in this release.

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References277

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
137,943,030Correction: Distinct neurexin-cerebellin complexes control AMPA- and NMDA-receptor responses in a circuit-dependent manner.Elife2023https://conductscience.org/library/papers/37943030PubMed
234,525,970NRXN1α+/- is associated with increased excitability in ASD iPSC-derived neurons.BMC Neurosci2021https://conductscience.org/library/papers/34525970PubMed
331,566,781Structures of neurexophilin-neurexin complexes reveal a regulatory mechanism of alternative splicing.EMBO J2019https://conductscience.org/library/papers/31566781PubMed
417,989,066Comparative genome hybridization suggests a role for NRXN1 and APBA2 in schizophrenia.Hum Mol Genet2008https://conductscience.org/library/papers/17989066PubMed
534,171,098Neurodevelopmental Disorders, Glycemic Control, and Diabetic Complications in Type 1 Diabetes: a Nationwide Cohort Study.J Clin Endocrinol Metab2021https://conductscience.org/library/papers/34171098PubMed
631,784,728Neuronal impact of patient-specific aberrant NRXN1α splicing.Nat Genet2019https://conductscience.org/library/papers/31784728PubMed
729,997,391Exome sequencing in congenital ataxia identifies two new candidate genes and highlights a pathophysiological link between some congenital ataxias and early infantile epileptic encephalopathies.Genet Med2019https://conductscience.org/library/papers/29997391PubMed
817,035,546Important contribution of alpha-neurexins to Ca2+-triggered exocytosis of secretory granules.J Neurosci2006https://conductscience.org/library/papers/17035546PubMed
932,709,898Atypical visual processing in a mouse model of autism.Sci Rep2020https://conductscience.org/library/papers/32709898PubMed
1035,831,437Autism-associated mutation in Hevin/Sparcl1 induces endoplasmic reticulum stress through structural instability.Sci Rep2022https://conductscience.org/library/papers/35831437PubMed

1 to 10 of 277

See all 277 references

Cited by5

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 4 citations.

PMIDTitleJournalYearIn PapersSource
42,971,955Sexually dimorphic control of excitatory synapse maintenance in ventral hippocampal circuits by Neurexin2α ligand Vstm2b.Nat Commun2026https://conductscience.org/library/papers/42971955PubMed
42,882,499The Role of the Immunoglobulin Superfamily of Cell Adhesion Molecules in Intellectual Disability.Cells2026https://conductscience.org/library/papers/42882499PubMed
42,737,512Modeling NDD-Associated NLGN2 Depletion Using CRISPR/Cas13 Reveals Exaggerated Process Elongation Mediated by the CCDC88A-G Protein-ELMO Axis.Int J Mol Sci2026https://conductscience.org/library/papers/42737512PubMed
42,589,270Signaling Through Girdin Underlies Excessive Cell Morphogenesis Resulting from Depletion of Neurodevelopmental Disorder-Related Neurexin-2.Int J Mol Sci2026https://conductscience.org/library/papers/42589270PubMed
41,998,081Neurexins, Ephrin receptors, and N-cadherin signaling as emerging mechanisms in synaptic dysfunction and neurodegenerative diseases.Commun Biol2026https://conductscience.org/library/papers/41998081PubMed

Genes1

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

GeneNCBI Gene IDLinked byIn Genes
IGSF2184,966PubTator's text mininghttps://conductscience.org/library/genes/84966

Source NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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